Explore chapters and articles related to this topic
Imaging flow cytometry for quantification of cellular parameters
Published in Raquel Seruca, Jasjit S. Suri, João M. Sanches, Fluorescence Imaging and Biological Quantification, 2017
Lymphocytes and monocytes can also adopt a different morphology depending on their phenotype or activation status. For example, T-lymphocytes stimulated with chemokines suffer shape alterations. Thus, imaging flow cytometry has been applied to correlate morphological changes induced by CCL19 with polarization of actin and phosphorylated Ezrin–Radixin–Moesin (ERM) [24] and to show that binding of oversulfated chondroitin sulfate (OSCS) to SDF-1α inhibited chemokine-induced morphological changes [25].
Exercise and inflammation in coronary artery disease: A systematic review and meta-analysis of randomised trials
Published in Journal of Sports Sciences, 2020
Gareth Thompson, Gareth W. Davison, Jacqui Crawford, Ciara M. Hughes
A summary of the results for each of the included studies is presented in ESM 1, Tables S1, S2, S3, and S4. Across the outcomes that could not be meta-analysed, beneficial within-exercise group changes were demonstrated for soluble tumour necrosis factor- alpha receptor 1 (TNF-α SR1) (P < 0.001) (Balen et al., 2008), chemokine (C-C motif) ligand 21 (CCL21) (P < 0.05) (Munk et al., 2011), and IL-35 (P = 0.001) (Raygan et al., 2017). Also, positive differences between exercise and control groups were observed for TNF-α SR1 (P = 0.004) (Balen et al., 2008), IL-35 (P = 0.002) (Raygan et al., 2017) and interferon gamma-induced protein 10 (IP-10) (P = 0.03) (Fernandes et al., 2011). No significant changes were reported within or between-groups for IL-33 (Raygan et al., 2017), monokine induced by gamma interferon (Mig) (Fernandes et al., 2011), monocyte chemoattractant protein 1 (MCP-1) (Munk et al., 2011), chemokine (C-C motif) ligand 19 (CCL19) (Munk et al., 2011), chemokine (C-X-C motif) ligand 16 (CXCL16) (Munk et al., 2011), CD40 ligand (CD40 L) (Munk et al., 2011), and pentraxin 3 (PTX-3) (Munk et al., 2011).