China 
Africa 
Explore chapters and articles related to this topic
What is “environmental illness”?
Published in Herman Staudenmayer, Environmental Illness, 2018
Multi-system symptoms are not unique to EI patients. They are mediated by the physiological systems involved in the human stress-response, presented in detail in Chapter 9. Somatization, anxiety, and psychophysiological disorders are associated with several psychiatric disorders described in Chapters 10, 11, and 12. A consistent finding in psychiatric epidemiology is that risk of psychiatric disorder increases linearly with the number of patient complaints (Goldberg and Huxley, 1970; Howard and Wessely, 1993). The severity of both current and lifetime psychiatric disorders correlates highly with the number of somatization symptoms (Russo et al., 1995). Central nervous system complaints (e.g., mental confusion, difficulty concentrating, poor memory, irritability, and mood alterations) are universal among EI patients. Fatigue is also typical, especially in EI patients with comorbid chronic fatigue syndrome (CFS) or “sick building syndrome” (Witorsch and Schwartz, 1994). Headache is the most common pain symptom. General arthralgia (aching joints) and myalgia (muscle aches) are also common and can lead to a diagnosis of fibromyalgia.
Reduction and Fixation of Sacroiliac joint Dislocation by the Combined Use of S1 Pedicle Screws and an Iliac Rod
Published in Kai-Uwe Lewandrowski, Donald L. Wise, Debra J. Trantolo, Michael J. Yaszemski, Augustus A. White, Advances in Spinal Fusion, 2003
Kai-Uwe Lewandrowski, Donald L. Wise, Debra J. Trantolo, Michael J. Yaszemski, Augustus A. White
Two types of lumbar spinal degeneration processes were identified: cases with severe disc degeneration and osteoarthritis of facet joints and those with osteoarthritis of the facet joints despite less severe disc degeneration. Most of the latter cases were males and were accompanied by painful arthralgia of the extremities.
Case series: rheumatological manifestations attributed to exposure to Libby Asbestiform Amphiboles
Published in Journal of Toxicology and Environmental Health, Part A, 2018
Roger Diegel, Brad Black, Jean C. Pfau, Tracy McNew, Curtis Noonan, Raja Flores
The patient was first seen at CARD in 2002 and then again in late-2009 when he reported breathing trouble over the last year or two when singing, and occasional sharp pains on the side of his chest. He was diagnosed with bilateral pleural thickening and sub-pleural interstitial changes. He was most recently seen at CARD in early-2014 and reported feeling somewhat better due to weight loss but continues with respiratory symptoms including chronic cough. His pulmonary function remained stable from 2002 to 2014. His most recent PFT in 2017 showed FVC 88%, FEV1 92%, and DLCO 117%. The patient was diagnosed with CREST variant of systemic sclerosis. The symptoms began with arthralgias in 1991 and 1992. The arthralgias continued and the patient saw a rheumatologist. A work up included X-rays of his hands, which were normal, no erosive disease. ANA test of 1:640 with negative SSA, negative SSB, negative double stranded DNA antibody, negative anti-Smith antibody, negative RNP antibody, negative rheumatoid factor, negative Lyme. A right forearm nodule was biopsied and revealed a subcutaneous granuloma, most consistent with a rheumatoid nodule. Sub-acute granuloma anulare was also considered in the differential. The patient was placed on hydroxychloroquine without any relief of the arthralgias. The patient was at that time diagnosed with undifferentiated connective tissue disease (UCTD). He started developing severe fatigue in August 1996, and Raynaud’s symptoms in July 1998. In 2000 and 2001, he began developing nodules on his forearms, thighs, lower extremities, hands, and fingers. Biopsy revealed multicentric reticulohistiocytosis. The symptoms continued, the Raynaud’s worsened, and he began developing sclerodactyly in his fingers, loss of pulp in the digits of his fingers and developed sclerodermatous facial features. He was diagnosed with a CREST variant of systemic sclerosis in March 2006. An ANA test in November 2001 had a titer greater than 1:320, and in January 2002, ANA titer was greater than 1:320. No reported ENA values.