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Vascular Anatomy
Published in Swati Goyal, Neuroradiology, 2020
The artery of Percheron is an anatomical variant of the posterior circulation in which a single common trunk arises from the first segment of either the left or right posterior cerebral artery (P1), that supplies the thalamus and midbrain bilaterally. Risk factors include hypertension, diabetes, and atrial fibrillation, which result in infarction secondary to occlusion of the artery of Percheron, due to atherosclerosis or embolism.
Etienne
Published in Walter J. Hendelman, Peter Humphreys, Christopher R. Skinner, The Integrated Nervous System, 2017
Walter J. Hendelman, Peter Humphreys, Christopher R. Skinner
The two PCAs originate at the termination of the basilar artery at the top of the brainstem; the PCA on each side runs back to the parietal and occipital lobes but also serves the medial and inferior portions of the temporal lobe. Close to its origin, the PCA gives off small branches to the medial thalamus (Figures 8.5 and 8.6). There is a forked artery close to the origin of the PCAs called the artery of Percheron, which has a single origin and serves the medial surface of both thalami adjacent to the third ventricle. Occlusion of this artery can lead to a medial thalamic syndrome characterized by apathy and anhedonia (inability to experience emotional pleasure), similar to a frontal lobe syndrome.
Vertical One-and-a-Half Syndrome with Pseudoabducens Palsy and Midbrain Horizontal Gaze Paresis
Published in Journal of Binocular Vision and Ocular Motility, 2022
Yasser Aladdin, Bader Shirah, Khurshid Khan
This case presented a triad of three distinct syndromes of horizontal gaze paresis, vertical one-and-a-half syndrome, and pseudoabducens palsy due to damage of a mixture of nuclear and supranuclear projections within the rostral mesencephalon. This combination was due to a single embolic infarct in the territory of the posterior thalamosubthalamic artery (artery of Percheron) that arises at the basilar bifurcation. Coexistence of these phenomena exemplified how rostral midbrain lesions may affect ocular motility in the vertical, horizontal, and torsional planes, along with disruption of normal vergence control.