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A brief history of dreams
Published in Josie Malinowski, The Psychology of Dreaming, 2020
This is very interesting in light of Mark Solms’s brain damage findings that we saw earlier. Remember how the individuals with brain damage who reported a loss of dreaming had damage to the PTO junction? This area has similarities with the ‘posterior hot zone’ (although the PTO junction is deeper in the brain). This area, then, when damaged, leads to a loss of dreaming, and when active, very accurately predicts dreaming. So far, then, this parietal-temporal-occipital area seems like a good contender for a neural correlate of dream generation.
Neurosurgery: Supratentorial tumors
Published in Hemanshu Prabhakar, Charu Mahajan, Indu Kapoor, Essentials of Geriatric Neuroanesthesia, 2019
Monica S. Tandon, Kashmiri Doley, Daljit Singh
The ST compartment primarily consists of the two cerebral hemispheres: diencephalon, basal ganglia (BG), ventricles, and the white matter (WM) tracts (Table 5.1). Anatomically, each cerebral hemisphere has five lobes—frontal, parietal, temporal, occipital, and insula. On basis of the cytoarchitecture (neuronal organization), the cerebral cortex is divided into 52 Brodmann areas; these areas closely correlate with the functional organization of the cerebral cortex. Functionally, the cerebral cortex is divided three categories: motor, sensory, and association areas (9).
The Problem and Clinical Methodology
Published in Aleksandr R. Luria, Lubov S. Tsvetkova, Robert J. Sbordone, Aleksandr Mikheyev, Sergei Mikheyev, The Neuropsychological Analysis of Problem Solving, 2017
Aleksandr R. Luria, Lubov S. Tsvetkova, Robert J. Sbordone, Aleksandr Mikheyev, Sergei Mikheyev
A patient with such lesions (most frequently localized to the parietal-temporal-occipital area in the left hemisphere) always remembers a problem given to him and works hard on analyzing the meaning of the statement. His intellectual activity retains its purposeful and selective character. The results of the operations are always compared with the original statement. The patient sees his mistakes and tries to correct them. In these cases, we witness, first and foremost, the impairment of the operations by which the patient attempts to solve the problem. For example, successful realization of intellectual processes is hindered precisely by impaired tactical strategies.
Clinicopathological Analysis of Sturge–Weber Syndrome with Focal Cortical Dysplasia FCD IIIc
Published in Fetal and Pediatric Pathology, 2023
Juan Cao, Guocheng Yang, Shoujun Xu, Pengyue Tang, Yue Wang, Yingying Shan, Yongxian Chen, Peng He
Global cortical atrophy (GCA) was assessed by preoperative magnetic resonance imaging (MRI) in all patients [7]. The fraction of atrophy in the frontal, parietal, temporal, occipital, and insula lobes in both hemispheres was summarized in this research. A score of 0 to 3 was given to each assessed area. No cortical atrophy equals a GCA grade of 0. Mild atrophy (opening of sulci) was a GCA grade 1. Moderate atrophy (volume loss of gyri) was a GCA grade 2, and severe atrophy (knife blade atrophy) was a GCA grade 3. The GCA score ranges from 0 to 15, from no atrophy to maximum atrophy.
The human functional connectome in neurodegenerative diseases: relationship to pathology and clinical progression
Published in Expert Review of Neurotherapeutics, 2023
Massimo Filippi, Edoardo Gioele Spinelli, Camilla Cividini, Alma Ghirelli, Silvia Basaia, Federica Agosta
Functional connectomics has also been employed to study atypical variants of AD. Posterior cortical atrophy patients compared to controls showed decreased small-worldness, as well as longer mean path length in parietal, temporal, occipital, sensorimotor areas and basal ganglia, both at the intra- and inter-hemispheric levels, even exceeding the pattern of structural and metabolic brain damage [33].
Freezing of gait in Parkinson’s disease: functional neuroimaging studies of the frontal lobe
Published in Neurological Research, 2018
María José Gallardo, Juan Pablo Cabello, María Jesus Corrales, Javier Torres-Donaire, Jose Javier Bravo, María Prado Talavera, Alberto León, Julia Vaamonde-Gamo
Lyoo and others [18] conducted a FDG PET study in PD patients with FOG who were treated with subthalamic nucleus deep brain stimulation (STN DBS). These authors found hypometabolism in parietal–temporal–occipital sensory association areas and concluded that this phenomenon limits the effectiveness of STN DBS in FOG patients.