Explore chapters and articles related to this topic
Osteoarthritis (OA)
Published in Charles Theisler, Adjuvant Medical Care, 2023
Spinal Manipulation: The Arthritis Foundation has recognized spinal manipulation as beneficial for spinal arthritis and recommends chiropractic care for spinal pain, stiffness, and limited motion.20 The American College of Physicians also recommends spinal manipulation, especially for low-back pain.21 Published research in the British Medical Journal by a rheumatologist summarized that the best treatment for cervical spondylosis, or neck arthritis, includes exercise and spinal manipulation.22
Etiology of RSD
Published in Hooshang Hooshmand, Chronic Pain, 2018
Cervical spondylosis is only matched by diabetes and syphilis as the “master immitators” in neurology. When dealing with headache, dizziness, shoulder pain, arm pain, or unexplained chest pain, cervical spondylosis should be considered at the top of the list of differential diagnosis.
Arthroscopic superior transverse scapular ligament release for suprascapular nerve compression at the suprascapular notch
Published in Andreas B. Imhoff, Jonathan B. Ticker, Augustus D. Mazzocca, Andreas Voss, Atlas of Advanced Shoulder Arthroscopy, 2017
Michael J. Messina, Marie Walcott, Michelle J. Chang, Jon J.P. Warner
SSN neuropathy may mimic symptoms of other shoulder pathologies or cervical spine diseases; as such, alternative diagnoses need to be ruled out before the diagnosis of SSN is considered in the differential diagnosis. SSN neuropathy may be associated with other shoulder pathologies such as labral tears and RCTs, so a high level of clinical suspicion is essential in order to avoid overlooking a potential source of pain from SSN compression. Patients frequently present with complaints of a dull, deep, aching type of pain that localizes to the posterolateral aspect of the shoulder and posterior scapula. This may be very similar to the presentation of cervical spondylosis, so a good examination of the cervical spine is essential. Night pain is common but not mandatory, and weakness with overhead activities and external rotation is frequently noted, but not essential to the diagnosis. There may be an associated history of repetitive overhead activities or trauma as well. Additional symptoms may be attributable to the presence of concomitant pathology, so the clinical picture can be confusing.
Anterior cervical discectomy and fusion is more effective than cervical arthroplasty in relieving atypical symptoms in patients with cervical spondylosis
Published in British Journal of Neurosurgery, 2022
Giovanni Grasso, Fabio Torregrossa, Brian A. Karamian, Jose A. Canseco, Alexander R. Vaccaro
Cervical spondylosis is a chronic and progressive disease of the cervical spine that serves as a common cause of cervical spine related dysfunction among adults.1 Cervical spondylotic disease is associated with intervertebral disc degeneration, disc herniation, ossification and hypertrophy of the posterior longitudinal ligament (PLL) and ligamentum flavum, osteophyte formation, central and foraminal stenosis, and instability. Together, these degenerative changes result in symptoms including neck and shoulder pain, numbness, hypersensitivity, difficulties with balance and gait, and impaired upper extremity fine motor skills.2 For patients with symptoms refractory to nonoperative management, surgery has been shown to be effective in improving clinical outcomes for patients with cervical spondylosis.3–15
Safety and feasibility of an early telephone-supported home exercise program after anterior cervical discectomy and fusion: a case series
Published in Physiotherapy Theory and Practice, 2021
Rogelio A. Coronado, Clinton J. Devin, Jacquelyn S. Pennings, Oran S. Aaronson, Christine M. Haug, Erin E. Van Hoy, Susan W. Vanston, Kristin R. Archer
Over the 6-month period, 29 consecutive patients were screened for inclusion into the case series. Twenty-one (72.4%) patients were excluded due to exclusion criteria (n = 7), declining participation (n = 1), canceling surgery (n = 1), incomplete baseline assessment (n = 1), not responding to study invitation (n = 8), and being unavailable during their preoperative clinic visit (n = 3). Eight (27.6%) patients were eligible, consented, and enrolled. These patients were predominantly female (63%), ranged in age from 35 to 77 years, were all married, and were mostly white (88%) (Table 2). Most patients (88%) had a diagnosis of cervical spondylosis (Table 3). The surgical indication included signs and symptoms of radiculopathy, myeloradiculopathy, or myelopathy. The number of cervical levels fused ranged from 1 to 3. Standard postoperative instructions from the treating surgeons included lifting restrictions (<15 lbs. for 6 weeks), no exercise or stretching involving sudden or extreme neck motion, advice to walk daily, and no driving for 2 weeks after surgery or within 24 hours after taking opioid pain medication. Recommendations for a cervical collar varied (Table 3). Valid physical activity data were obtained from seven (88%) patients at 6 weeks and 6 months.
A population-based case–control study of the association between cervical spondylosis and tinnitus
Published in International Journal of Audiology, 2021
Yen-Fu Cheng, Sudha Xirasagar, Tzong-Hann Yang, Chuan-Song Wu, Nai-Wen Kuo, Herng-Ching Lin
Cervical spondylosis is a chronic degenerative condition of the cervical spine that affects not only the vertebral bodies and the intervertebral discs in the neck, but also the facets, other joints and associated soft tissue supports. Progression of the spondylosis process may cause narrowing of the spinal canal and intervertebral formina, resulting in neurological deficits. Because the spinal cord integrates and relays somatosensory inputs, mechanical deformity caused by cervical spondylosis may interfere with the dorsal root ganglia (DRG) and dorsal column, from which sensory input to the central nervous system are relayed in the CN of the brainstem (Kelly et al. 2012). Disruption of the somatosensory pathway from the DRG or the dorsal column may trigger hyperactivity in the CN manifesting as tinnitus (Koehler and Shore 2013; Marks et al. 2018; Wu et al. 2016).