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Benign and Malignant Disease of the Oral Cavity
Published in R James A England, Eamon Shamil, Rajeev Mathew, Manohar Bance, Pavol Surda, Jemy Jose, Omar Hilmi, Adam J Donne, Scott-Brown's Essential Otorhinolaryngology, 2022
Leukoedema and nicotinic stomatitis: generalised white change of the oral mucosa. Most commonly seen in smokers. Sometimes red spots on white background when openings of minor salivary glands are visible.
Check the Cancer Before It Checks You Out
Published in Prakash Srinivasan Timiri Shanmugam, Understanding Cancer Therapies, 2018
Early detection of mucosal lesions can be enhanced by the use of a dilute acetic acid rinse and observation under a chemiluminescent light (ViziLite). In one study, 78 of 100 patients who presented for dental screening were examined by COE (under incandescent light). After a 1-minute rinse with 1% acetic acid and then using the ViziLite, 57 clinically diagnosable benign lesions (e.g., linea alba, leukoedema) and 29 clinically undiagnosable lesions were found initially. After the acetic acid rinse again, six additional diagnosable lesions (linea alba) and three undiagnosable lesions were found. In a multicenter study, increased visibility of lesions visible by COE was reported. In that and other studies, ViziLite revealed occasional lesions not seen under COE, but occasionally, the converse has been the case, so the jury again is out on the real benefits (Scully et al. 2008).
The Adverse Effects of Alcohol and Drug Abuse in the Oral Cavity
Published in John Brick, Handbook of the Medical Consequences of Alcohol and Drug Abuse, 2012
Terry D. Rees, Robert A. Levine
To date, the adverse intraoral and perioral effects of cannabis have not been adequately studied under controlled experimental conditions. However, numerous case reports have described xerostomia, nicotinic stomatitis, gingival and mucosal burns, erythematous enlargement of the uvula, leukoedema, traumatic ulcers, leukoplakia (white patches), overgrowth of gingival soft tissues, increased severity of gingivitis, and oral cancer (Arendorf, 1993; Baddour, Audemorte, and Layman, 1984; Baddour et al., 1984; Colon, 1972; Darling, 2003; Donald, 1986; Layman, 1978).
Intraoperative storage of saphenous vein grafts in coronary artery bypass grafting
Published in Expert Review of Medical Devices, 2019
Catherine J. Pachuk, Sophie K. Rushton-Smith, Maximilian Y. Emmert
The loss of viability in the ischemic HSV model and the endothelial cell damage observed in the PMV model are not entirely unexpected given the inability of saline to protect against ischemic damage. Saline may potentially also exert active ‘solution damage’, given its acidic pH (5.5) and lack of ionic balance. The potential for active damage may be substantiated by the number of reports on tissue damage attributed to saline [22–25,28–35], which are broad in scope and cover the harmful effects of saline as an irrigation, wetting, or tissue storage, as an intravitreal injection solution, as a resuscitation fluid, and even as a short-term holding solution for human biopsies prior to fixation. Sengupta et al report saline effects mimicking pathological findings including mild dysplasia, basilar degeneration and false appearance of leukoedema [21]. They concluded, ‘In no way should normal saline be used, even for a short time, to store and transport excised tissue; it is imperative that such tissues [for biopsy] be placed immediately in a proper fixative like 10% formalin’. Based on these observed detrimental effects, it should not be surprising that a large amount of literature also exists specifically citing saline-mediated damage of vascular grafts occurring after even a short-term exposure (10 minutes) [23–25].
Determination and diagnostic value of CA9 mRNA in peripheral blood of patients with oral leukoplakia
Published in Journal of Enzyme Inhibition and Medicinal Chemistry, 2018
Manuel Torres López, Mario Pérez Sayáns, Cintia Chamorro Petronacci, Francisco Barros Angueira, Pilar Gándara Vila, Alejandro Lorenzo Pouso, Abel García García
Exclusion:Patients with the clinical appearance or histological findings of white lesions, such as leukoedema, linea alba, lesions such as lichen planus, lupus erythematosus, etc.Patients with follow-up periods of less than 1 year.Patients for whom there are no clinical images of the lesions to be studied.Patients without initial histological diagnosis.Patients whose anatomopathological materials are not available to confirm the initial diagnosis.