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Motor Aspects of Lateralization: Evidence for Evaluation of the Hypotheses of Chapter 8
Published in Robert Miller, Axonal Conduction Time and Human Cerebral Laterality, 2019
The third syndrome to be dealt with here, conduction aphasia, was predicted by Wernicke, on the basis that there must be connections between Broca’s and Wernicke’s area, which could be damaged without damage to either of the cortical regions. Nevertheless, this variety of aphasia is not universally recognised, or its occurrence is said to be rare. Benson (1979) however, regards it as a distinct syndrome occurring in 10-15% of cases of aphasia. As a discrete entity this syndrome can be produced by relatively small lesions of the inferior parietal lobe. Lesions confined to Wernicke’s area can sometimes produce the same syndrome. For the inferior parietal lesions, Benson admits that empirically it is undecided whether these lesions need only affect the white matter bundle known as the arcuate fasciculus, or also require, as an essential component, damage to the cortical tissue in this region. According to Benson (1979), in conduction aphasia, comprehension is normal or almost so. Speech is fairly fluent, and is not excessive. It may have a broken, dysprosodic character, with pauses and hesitations, apparently for word finding. Paraphasias occur, as in Wernicke’s aphasia. There is however a serious problem in repeating spoken language, and the hallmark of conduction aphasia is the great difference between this and the almost intact comprehension. Patients who fail in a repetition test may convey the meaning of the sentence they are supposed to repeat by creating a paraphrase with similar meaning. In addition to repetition deficits, there is a deficit in confrontation naming.
Discussions (D)
Published in Terence R. Anthoney, Neuroanatomy and the Neurologic Exam, 2017
♦1. Is the term “central aphasia” synonymous with the term “global aphasia”?the term “receptive aphasia”?the term “conduction aphasia”?
Recognising and engaging with language problems
Published in Ross Balchin, Rudi Coetzer, Christian Salas, Jan Webster, Addressing Brain Injury in Under-Resourced Settings, 2017
Ross Balchin, Rudi Coetzer, Christian Salas, Jan Webster
Conduction aphasia typically results from damage to the parietal lobe and the underlying white matter. A stroke is frequently the cause, especially one that involves a blockage (embolus) of the brain’s middle cerebral artery (MCA).
Stuttering and compulsive manipulation of tools after hemorrhage in the anterior corpus callosum and cingulate gyrus: a case study
Published in Speech, Language and Hearing, 2018
The primary characteristics of this patient's disfluency were blocks with struggle and repetition of syllables during the first disfluency test (see Table 2). These characteristics distinguish this form of disfluency from any utterance-based disorder following brain injury such as conduction aphasia or palilalia. Conduction aphasia is characterized by frequent occurrence of phonemic paraphasia. The disfluency blocks and syllable repetitions observed in the current patient consisted of correct pronunciation of syllables, which differs from mistakes in the choice of syllables, i.e., phonemic paraphasia. Palilalia is a phenomenon whereby one repeats a word and a phrase involuntarily. It gradually becomes a rapid utterance along with a low voice. Palilalia also occurs often in sentence-final contexts and is absent from reading. However, our patient had neither a rapid utterance or a low voice. Therefore, the disfluency exhibited by this patient seems to be neither conduction aphasia nor palilalia.
Atypical and early symptoms of sporadic Creutzfeldt – Jakob disease: case series and review of the literature
Published in International Journal of Neuroscience, 2021
Grammatiki Katsikaki, Ioannis E. Dagklis, Petros Angelopoulos, Dimitrios Ntantos, Angeliki Prevezianou, Sevasti Bostantjopoulou
Cognitive impairment is a fundamental characteristic of CJD [13] and mutism is observed in many patients during the course of the disease [6]. Acquired language, communication and writing impairments, like aphasia and agrafia, are rarely isolated clinical features during the early phase of the disease (∼1%) [112]. Aphasia appears mainly as anomic, non-fluent, logopenic and may be inconsistent with apraxia [16,113–120]. Conduction aphasia has been described as well [121]. These symptoms are associated with malfunction mainly of the Sylvian fissure cortex of the dominant hemisphere, but also of the frontal, temporal or parietal lobe [114,115,117,121].
Analysing coherence of oral discourse among Cantonese speakers in Mainland China with traumatic brain injury and cerebrovascular accident
Published in International Journal of Speech-Language Pathology, 2020
Anthony Pak-Hin Kong, Dustin Kai-Yan Lau, Chloe Yuen-Yi Cheng
A total of 36 individuals participated in this study (see Table I). They included 18 speakers with fluent aphasia, seven of which were induced by CVA and 11 induced by TBI, as well as 18 NBI control participants. All were recruited from the Guangdong Work Injury Rehabilitation Hospital, with the TBI or CVA diagnosed by neurologists and/or medical internists (see Table I for neuroimaging findings), and were native speakers of Cantonese who were born in the Guangdong province of Mainland China. None of the participants received formal language interventions that addressed the coherence and/or cohesion of spoken discourse. However, prior to the time this study was conducted, they had received training focussing on swallowing (primarily) and functional communication, with or without group interventions mediated by occupational therapists and/or physiotherapists. In other words, the treatment goals of these participants did not overlap with the aims of the present study and should not have any influence on the results. All participants in the TBI group had experienced a closed head brain injury that occurred at least six months prior to testing and were diagnosed with anomic aphasia with the Cantonese version of the Western Aphasia Battery (CAB; Yiu, 1992). As for the CVA group, all had experienced a cerebrovascular accident that had occurred at least one month prior to testing. Based on the CAB, six of them were diagnosed with anomic aphasia and one with conduction aphasia. While Yiu (1992) did not specify the ranges of aphasia quotients (AQ) for various aphasia severity, only participant LYF with Conduction aphasia demonstrated a lower AQ of 60.8, clinically judged as moderate aphasia. The remaining participants had a mild-to-moderate or mild anomic aphasia.