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Coxiella
Published in Dongyou Liu, Handbook of Foodborne Diseases, 2018
Other chronic Q fever manifestations include spontaneous abortion or premature labor (likely due to placentitis), thyroiditis, mediastinal lymphadenopathy, pancreatitis, mesenteric panniculitis, epididymitis, orchitis, priapism, syndrome of inappropriate antidiuretic hormone (SIADH) secretion, optic neuritis, Guillain-Barré syndrome, interstitial fibrosis, pseudotumor, glomerulonephritis, osteomyelitis, coxitis, spondylodiskitis, arthritis, septic arthritis, chronic fatigue syndrome, and TORCH syndrome (toxoplasmosis, other infections, rubella, cytomegalovirus infection, and herpes simplex) [30].
Fetal, Perinatal and Infant Autopsies
Published in Julian L Burton, Guy Rutty, The Hospital Autopsy, 2010
Whereas most situations would usually require some degree of microbiological study, fetuses or infants more strongly suspected of having infection should be more rigorously investigated and further sampling should be carried out. Clinical situations indicating a strong likelihood of infection (bacterial or viral) include: maternal vaginal discharge;discoloured or malodorous placenta and/or membranes;premature prolonged rupture of membranes;maternal pyrexia, before or after delivery;fetal skin lesions;intrauterine growth retardation;TORCH syndrome (toxoplasmosis, other infection, rubella, cytomegalovirus, herpes simplex);non-immune hydrops (usually viral aetiology);following a period of intensive care (especially preterm infants).
Relative Expression of OX40, OX40L mRNA, and OX40L Serum Levels in Women with Recurrent Spontaneous Abortion
Published in Immunological Investigations, 2019
Fateme Rahmani, Hossein Hadinedoushan, Nasrin Ghasemi
This case-control study was carried out on two different groups who referred to Yazd Reproductive Sciences Institute, Iran. The case group consisted of 40 women with a history of two or more primary RSAs who were at least 3 months past their abortion. The control group consisted of 40 non-pregnant women in gestational age that had no abortion history and had at least one successful pregnancy. The age was matched between the two groups. All cases were examined by a gynecologist. Inclusion criteria were: no male factor causes, no anatomical and endocrinal abnormalities, normal parental karyotyping, TORCH syndrome-negative, no thrombophilia, absence of anti-phospholipid antibodies (IgM and IgG classes), anti-cardiolipin antibodies (IgM, IgG and IgA classes), and anti-nuclear antibodies (IgM and IgG classes). We also checked serum thyroid hormones, anti-thyroid peroxidase, and anti-thyroglobulin. Moreover, those suffering from thyroid disorders were excluded from the study. Five milliliters of peripheral blood were collected and information on variables such as age, number of abortions, and number of pregnancies was provided. The Ethics Committee of Shahid Sadoughi University of Medical Sciences, Yazd, Iran, approved the research protocol. Informed written consent was obtained from all the enrolled participants.
Enhanced S-Cone Syndrome Masquerading as TORCH in an Infant and a Toddler
Published in Ocular Immunology and Inflammation, 2023
Ana Navarrete, Milka Matanis-Suidan, Itzhak Hemo, Hadas Mechoulam, Eyal Banin, Radgonde Amer
Pseudo-Torch syndrome is an autosomal recessive condition, clinically similar to the congenital TORCH infection, however, in the absence of an infectious agent.2 It was first described by Baraitser et al.2 who reported two brothers with intracranial calcification associated with microcephaly from birth, spastic tetraparesis, and epilepsy. Similar cases have been subsequently reported in literature.3–5
Congenital cytomegalovirus infection: epidemiology, prediction, diagnosis, and emerging treatment options for symptomatic infants
Published in Expert Opinion on Orphan Drugs, 2020
Nobuhiko Nagano, Ichiro Morioka
Congenital CMV infection, long known as a disease of TORCH syndrome, is a CMV infection transmitted from the mother to the fetus via the placenta and may or may not cause any symptom [6,7]. Antiviral therapies for infants with symptomatic congenital CMV infection have recently gained much attention and have been demonstrated to be well tolerated and effective in improving hearing and neurodevelopmental outcomes [8–13].