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Prevention of pre-eclampsia
Published in Pankaj Desai, Pre-eclampsia, 2020
There is still more to this. By understanding the difference between derangement and disease, one can predict if these conditions will be cured or will be controlled. Diseases get cured, and derangements get controlled. Nutritional anaemia in pregnancy is a disease. It can be cured by administering the nutritional hemopoietic substance. On the other hand, non-nutritional anaemia resulting from sickle cell disease or thalassemia is a derangement. Therefore, these conditions cannot be cured. They can either be controlled or if the entire marrow is replaced can be changed, but they cannot be cured. Diabetes too is a derangement. It is a derangement of insulin production, activity and metabolism. The basic cause is insulin deficiency which may be genetic or age acquired. Therefore, diabetes can be controlled, but it cannot be cured. Diabetes is, therefore, not a disease it is a derangement.
Nutraceuticals and Anaemia in Pregnancy
Published in Priyanka Bhatt, Maryam Sadat Miraghajani, Sarvadaman Pathak, Yashwant Pathak, Nutraceuticals for Prenatal, Maternal and Offspring’s Nutritional Health, 2019
Anaemia can be classified by the cause into nutritional anaemias and haemolytic anaemias. It can also be classified according to variation in the colour, sizes, and shapes of the red blood cells. The size of the red blood cells is smaller than normal in microcytic anaemias, e.g. iron-deficiency anaemia, while the red blood cells are larger in size than normal in macrocytic anaemia, e.g. megaloblastic anaemia (Figure 7.2). In iron-deficiency anaemia, there is deficiency of iron resulting in a decreased amount of haemoglobin in each red blood cell, while in megaloblastic anaemia, there is deficiency of folate or vitamin B12 (De-Regil et al. 2014).
The Problems of Undernutrition
Published in R. J. Jarrett, Nutrition and Disease, 1979
Nutritional anaemias comprise the second most common group of deficiency disorders after protein-calorie malnutrition. Nutritional anaemia is defined as anaemia which occurs when there is a deficiency of one or more of the essential nutrients required for the synthesis of haemoglobin and the production of erythrocytes. Several nutrients are required for erythropoiesis.
Anemia in Sri Lanka: A Literature Review
Published in Hemoglobin, 2022
A. A. Dinusha S. Amarasingha, H. J. Ruwindi L. Silva, P. Shiromi Perera, Anuja P. Premawardhena
The systematic search was conducted in MEDLINE via PubMed using the following search terms: anemia, iron deficiency anemia and nutritional anemia, malaria and parasitic infections, anemia in chronic diseases, anemia in malignancies, hemoglobinopathies, red cell enzymopathies, red cell membranopathies, and Sri Lanka in many combinations. The articles that were published in English were included. The systematic search was conducted from January 2020 to May 2021. Separate searches were done in the OVID and EMBASE databases using similar methods. Missing articles were checked using the “similar articles” facility in PubMed and by the citations of previous reviews. All articles were reviewed by two authors independently. Final decision on selection was made via consensus. All articles were reviewed by a third independent author. Data on the sample size, sample design, study setting, area, study period, age group, and research findings of all the selected articles were extracted.
Traditional green leafy vegetables as underutilised sources of micronutrients in a rural farming community in south-west Nigeria II: consumption pattern and potential contribution to micronutrient requirements
Published in South African Journal of Clinical Nutrition, 2021
Shirley Isibhakhomen Ejoh, Faustina Dufie Wireko-Manu, David Page, Catherine MGC Renard
In Nigeria, apart from under-nutrition and over-nutrition, which currently constitute a double burden of disease, hidden hunger also constitutes a third burden.1 Vitamin A deficiency (VAD) and iron deficiency remain public health challenges in Nigeria and this is particularly so in rural areas of the country. Some 25% of infant, child and maternal mortality is attributed to vitamin A deficiency.1 Prevalence of vitamin A deficiency among children aged under five children in Nigeria is between 5.3% and 29.5%,2,3 depending on the region. Iron deficiency is the cause of prevalent nutritional anaemia (also known as iron deficiency anaemia) in Nigeria; 20–40% in adult females, 20–25% in children and 10% in adult males.1 High rates of micronutrient deficiency are an indication of low-quality diets, which also contributes to all other forms of malnutrition.4 The micronutrient quality of poor diets could be improved with readily available and accessible traditional green leafy vegetables (TGLVs), especially among populations who have inadequate access to animal source foods for economic reasons.5
Comparison of the efficacy of parenteral and oral treatment for nutritional vitamin B12 deficiency in children
Published in Hematology, 2018
Rabia Gönül Sezer, Handan Ayhan Akoğlu, Abdulkadir Bozaykut, Gül Nihal Özdemir
In a study conducted by Chandelia et al., children with nutritional anemia (n = 150) were randomized to receive iron, folic acid or iron, folic acid and cobalamin [12]. Cobalamin levels were tested in 41 children and 97.5% had cobalamin deficiency. Cobalamin was administered as either oral tablets daily or intramuscular injections for the treatment. Treatment protocols were as follows; oral group: 125 μg, 250 μg and 500 μg for children under 1 y, 1–2 y or older than 2 y, respectively; parenteral group: 250 μg or 500 μg for children younger than 2 y and 2–5 y respectively; on days 1, 15 and 29. At 2, 4, and 8 weeks, hemoglobin levels were measured and higher hemoglobin levels were observed in the cobalamin group. However, there was no statistically significant difference between oral and parenteral arms [12]. The authors reported that oral administration is as effective as intermittent parenteral administration [12]. Verma et al. treated 28 children aged between 6 months and 18 years with macrocytic anemia [14]. Oral treatment consisted of 500 mcg tablet of methylcobalamin once daily at a dosage of 30 mcg/kg/day [14]. The authors demonstrated the hematological and biochemical response to oral vitamin B12 therapy at 1 month with a mean increase in hemoglobin, absolute reticulocyte count, platelets, holotranscobalamin and vitamin B12 levels, also with mean fall in mean corpuscular volume [14]. Post-treatment correction of vitamin B12 levels was observed in all the children [14].