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Cyanotic congenital heart diseases in adulthood
Published in Jana Popelová, Erwin Oechslin, Harald Kaemmerer, Martin G St John Sutton, Pavel Žáček, Congenital Heart Disease in Adults, 2008
Jana Popelová, Erwin Oechslin, Harald Kaemmerer, Martin G St John Sutton, Pavel Žáček
The following parameters should be monitored regularly: blood count with special emphasis on hematocrit, erythrocyte count and indices, platelet counts, iron, ferritin, coagulation parameters, uric acid, urinalysis, ureate, creatinine, aspartate aminotransferase (ASAT), alanine aminotransferase (ALAT), bilirubin, gamma glutamyltranspepsidase (GGT), alkaline phosphatase (ALP), total protein and albumin. Hyperbilirubinemia and increased GMT are early markers of congestive hepatopathy. Ferritin is an early and sensitive marker of reduced iron stores if its serum levels fall to <15µg/l. However, ferritin levels tend to rise in chronic infectious and inflammatory diseases irrespective of iron stores. Microcytosis develops in cases of chronic iron deficiency. Hyperhomocysteinemia due to folate or vitamin B12 deficiencies may mask iron deficiency.10,23
A simple discharge risk model for predicting 1-year mortality in hospitalised acute decompansated heart failure patients with reduced ejection fraction
Published in Acta Cardiologica, 2018
Kurtulus Karauzum, Irem Karauzum, Dilek Ural, Canan Baydemir, Mujdat Aktas, Umut Celikyurt, Guliz Kozdag, Onur Argan, Serdar Bozyel, Aysen Agir
The presence of hepatomegaly at admission was found to be a predictor of mortality in the study population. Hepatic dysfunction is caused by impaired hepatic circulation in terms of congestion and/or hypo perfusion. Congestive hepatopathy is more common and it reflects excessive peripheral congestion [16]. Hepatomegaly is a significant finding of right sided HF. The presence of hepatomegaly in the HFrEF indicates that the disease is accompanied by right sided HF which is compatible with advanced disease. In fact, passive congestion secondary to right-sided HF and reduced arterial perfusion and oxygenation due to left-sided HF often coexist and potentiate the deleterious effects of each other on the liver [17]. Previous studies and registries have demonstrated the importance of hepatomegaly in predicting rehospitalization and mortality [18–20].
Parameters affecting outcome of paediatric cardiomyopathies in the intensive care unit: experience of an Egyptian tertiary centre over 7 years
Published in Libyan Journal of Medicine, 2020
Alaa A. Sobeih, Sonia A. El-Saiedi, Noha S. Abdel Khalek, Shereen A. Attia, Baher M. Hanna
The abnormality in levels of transaminases indicates cardiac hepatopathy and was associated with mortality in our study. This finding might indicate congestive hepatopathy and/or acute cardiogenic liver injury are associated with circulatory failure. In chronic heart failure, cell death is due to apoptosis whereas necrotic cell death is prominent in acute heart failure [40]. AST, together with cholestasis markers including γ-glutamyl-transpeptidase, total bilirubin and alkaline phosphatase were correlated with poor outcome in congestive hepatopathy, while ALT and lactate dehydrogenase were important markers in acute cardiogenic liver injury [41].
Giant tumour thrombus with congestive hepatopathy: value of multimodality imaging
Published in Acta Cardiologica, 2022
Physical examination showed caput medusa and peripheral oedema. Liver tests suggested congestive hepatopathy (i.e. elevated gamma-glutamyltransferase and alkaline phosphatase). Echocardiography subcostal view revealed a mass in the right atrium bulging from the inferior vena cava (IVC) (*, Figure 1(A), Supplementary data online, video S1 and S2).