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Organophosphorous Compounds
Published in Fina P. Kaloyanova, Mostafa A. El Batawi, Human Toxicology of Pesticides, 2019
Fina P. Kaloyanova, Mostafa A. El Batawi
Rosin analyzed 222 case histories of persons intoxicated by methylmercaptophos and established that the dermal route of penetration was the most probable compared to inhalatory or oral penetration, it had a longer latent period (3 to 8 h).145 The earliest symptoms of intoxication are giddiness with blurred vision, a headache located mainly in the parietal-temporal area, and general weakness. Later on nausea, vomiting, pains in the epigastric and abdominal area, and diarrhea appear. Pallor of the skin and membranes and furred tongue are observed, as well as moderate tachycardia and less often bradycardia, dull cardiac sounds. Single crepitations are detected in the lungs. Better expressed are the changes in the nervous system — apathy and sleepiness or on the contrary, excitement and irritativeness. Almost all sufferers show a moderate sweating, cooling extremities, and cyanosis of fingers and toes. In more severe intoxications, these symptoms are better expressed.
Environmental contaminants and preeclampsia: a systematic literature review
Published in Journal of Toxicology and Environmental Health, Part B, 2018
Emma M. Rosen, MG Isabel Muñoz, Thomas McElrath, David E. Cantonwine, Kelly K. Ferguson
The clinical definition and diagnosis of preeclampsia has changed relatively little in the last 60 years and is primarily characterized by new onset or worsening hypertension (blood pressure greater than or equal to 140 mm Hg systolic or 90 mm Hg diastolic). Prior to 2013, new-onset significant proteinuria at or after 20 weeks’ gestation (excretion of ≥300 mg of protein in urine during a 24-h urine collection, or protein/creatinine ratio ≥ 0.3) was required for diagnosis. However, the definition has expanded so that in combination with hypertension, any of the following (at or after 20 weeks’ gestation) are sufficient for diagnosis: new-onset thrombocytopenia (platelet count < 100,000/microliter), impaired liver function (abnormally elevated blood concentrations of liver enzymes and/or severe persistent right upper quadrant or epigastric pain), renal insufficiency (serum creatinine ≥ 1.1 mg/dl or a doubling of the serum creatinine concentration), pulmonary edema, or visual or cerebral disturbances (ACOG 2013).
Visfatin gene expression and oxidative stress in pregnancy induced hypertension
Published in Egyptian Journal of Basic and Applied Sciences, 2018
Hanan M.A. El-Taweel, Nevein A. Salah, Amal K. Selem, A.A. El-Refaeey, A.F. Abdel-Aziz
The exact reasons of preeclampsia are not well known so risk of it may be elevated by some factors such as the first pregnancy [5]. Also women with a history of preeclampsia [4] and multiple gestations elevate the risk [2]. Furthermore, some disease present before pregnancy such as obesity, diabetes mellitus, insulin resistance, chronic hypertension, gestational diabetes, lupus, vascular or connective tissue disorders and also chronic kidney disease may be increase the risk of preeclampsia [6]. High blood pressure during pregnancy is divided into four groups as recommended by the National High Blood Pressure Education Program Working Group on High Blood Pressure in Pregnancy. The first group is chronic hypertension, the second group is gestational hypertension, the third group is mild and severe preeclampsia and the fourth group is preeclampsia superimposed on chronic hypertension [7]. Gestational hypertension and preeclampsia generally can be diagnosed by measurements of high blood pressure and proteinuria [8]. Also some symptoms may be associated with increasing in blood pressure such as vision interruption, continuous acute headaches, unexpected expand in face, hands and feet, vomiting, epigastric pain, lowering in platelets and increasing in liver enzymes and serum creatinine [9].
Investigation of Metal levels in Artemisia herba-alba Medicinal Plant and Soil Samples Collected from Different Areas in Jordan Country
Published in Soil and Sediment Contamination: An International Journal, 2021
Adnan M. Massadeh, Abdulwahab O. El-Rjoob, Mohammad N. Omari
For example, Ni affects on different organs such as kidneys, lungs, and liver, and produces multiple toxic effects such as decreased lung function, heart attack, kidney damage, low blood pressure (Das, Das, and Dhundasi 2008; Guo et al. 2014; Munoz and Costa 2012; Schaumlöffel 2012), whereas, Cu toxicity is associated with nausea, vomiting, and epigastric pain (Nolan 2003; Olivares 1996).