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Diabetes Mellitus, Obesity, Lipoprotein Disorders and other Metabolic Diseases
Published in John S. Axford, Chris A. O'Callaghan, Medicine for Finals and Beyond, 2023
Hypothalamic autonomic centres are activated by blood glucose concentrations ≤2 mmol/L and stimulate both sympathetic and parasympathetic pathways; the resulting release of catecholamines, cortisol, glucagon and growth hormone raises blood glucose concentrations. Autonomic activation produces symptoms of cold sweat, tachycardia, tremor, blurred vision, hunger and altered salivation.
March 2006–May 2007
Published in Kate Hayward, From Oncology Nursing to Coping with Breast Cancer, 2021
I woke up at 3 am in a cold sweat. Felt horrible but not sick. Phil brought me ginger biscuits and anti-emetics before he went to work. Thankfully I managed to get back to sleep. Spent most of the day writing cards and on the phone to friends. Very difficult finding the right way to tell them. Rang the Robert Ogden Macmillan Centre at St James’s to make HAT appointment (Head Art Therapy), in case the cold cap doesn’t work and I need a wig and scarves.
Ectopic Pregnancy: Extrauterine Pregnancy and Pregnancy of Unknown Location
Published in Botros Rizk, A. Mostafa Borahay, Abdel Maguid Ramzy, Clinical Diagnosis and Management of Gynecologic Emergencies, 2020
In case of a tube rupture, which usually occurs only if the EP is located in the isthmus of the fallopian tube, stage I and II are skipped and the symptoms occur immediately. The symptoms might be similar to those of shock, such as tearing pain, cold sweat, a sensation of weakness, collapse, and dyspnea. The patient may experience heavy intra-abdominal bleeding. The abdomen is then very sensitive to pressure. A vaginal examination in this situation is usually not tolerated by the patient.
Trick Mirrors: The Shape-Shifting of White CIS-Women as Survivors / Perpetrators / Comrades in the Fight for Bodily Autonomy
Published in Studies in Gender and Sexuality, 2023
About five years ago, I awoke in a cold sweat. I had had a dream that culminated in me sitting on the curb of a familiar street, feeding some kits avocados. My perspective was zoomed in, and I was watching myself hand-feed the little mouths. Yet, despite this moment that felt like it should be precious, I felt dread and disgust throughout my body. I couldn’t understand what I felt, it felt so discordant with the scene, but as my perspective expanded and I took in a wider view, I saw that the baby foxes had no bodies. Their disembodied heads eagerly lapped up the avocado and I felt my chest tighten. I looked away, unable to bear it, and that is when I saw the mother, trotting away. She paused, looking back at me over her shoulder from a distance, and I felt an uneasy sense of knowing: These kits could never survive without their bodies—wasn’t that why their mother was leaving? So why did I continue to feed them? And yet, the thought of withholding sustenance from the little heads shook me, and I seemingly could not deny their hunger.
Quercetin with lycopene modulates enzymic antioxidant genes pathway in isoproterenol cardiotoxicity in rats
Published in Libyan Journal of Medicine, 2021
LIJUAN Chen, XIAOLI Wu, WEIWEI Wang, XIA Wang, Jianhua Ma
Cardiovascular disease (CVD) is a group of disease that involves blood vessels of heart, brain, arms and legs. People with CVD are presented with various forms of complications such as atherosclerosis, heart failure and stroke. Normally, both heart failure and strokes are caused by a blockage of fatty deposits on the inner walls of blood vessels that supply blood to the heart or brain. Such blockage is associated with numerous behavioral activity such as unhealthy diet, lack of exercise, excessive alcohol and tobacco use as well as history of diabetic and hypertension (Turer, A.T. and Hill, J.A. 2010). Often, many alarming warnings of CVD are prominent, which includes numbness or pain in face and chest area, shortened breath, nausea, breaking into a cold sweat and loss of coordination. Manifestation of untreated CVD complications may lead to lethal outcomes in myocardial infarction (MI). Myocardial ischemia (MI) refers to an exacerbation of cardiac tissue injury due to decreased oxygen supply and/or decreased blood supply to cardiac tissues (Kocak et al., 2016).
Bilateral reactive and then fixed mydriasis as a marker of hypovolemic shock
Published in British Journal of Neurosurgery, 2020
Arash Fattahi, Abdoulhadi Daneshi
This has been reported in unstable hemodynamic situations caused by a ruptured cardiac wall.1 The authors considered the pupil abnormalities due to global cerebral ischemia.1 Although the final result of severe hypovolemia and hypoperfusion is irreversible brain damage, in early stages, dilated pupils may be a sign of sympathetic hyperactivation or hypoperfusion. In other reports, the authors concluded that dopamine administration could reverse hypovolemic shock-induced bilateral mydriasis.2 Although we did not prescribe dopamine for our patient, administration of it could relief hypoperfusion and resulting bilaterally dilated pupils, as a vasopressor and inotrope drug.3 Other conditions that may cause dilated pupils in the context of head injury are seizure, raised intracranial pressure and concomitant toxicity. Absence of cold sweat and no urine output in our case suggest at least a contribution from hypoperfusion.