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The Follow-Up Metabolic Medicine Hospital Consultation
Published in Michael M. Rothkopf, Jennifer C. Johnson, Optimizing Metabolic Status for the Hospitalized Patient, 2023
Michael M. Rothkopf, Jennifer C. Johnson
But there are a few exceptions that deserve special mention here. For the most part, the fat-soluble vitamins A, D, E and K have long enough half-lives that their deficiency may take months to develop. Even if deficiencies do develop, the acute effects of these losses are small. They cause chronic illness, rather than acute disease.
Inflammatory, Hypersensitivity and Immune Lung Diseases, including Parasitic Diseases.
Published in Fred W Wright, Radiology of the Chest and Related Conditions, 2022
Some patients with acute disease may be ill, very rarely with a fulminating life-threatening condition. The author remembers a 17 year old youth who presented with rapidly progressive acute erythema nodosum, acutely progressive breathlessness and early infiltration on a chest radiograph. He came from his GP, but as he looked so unwell, the author kept him in the radiology department, and then had him admitted to hospital. Later that day he had to be transferred to ITU, developed disseminated intravascular coagulation, but eventually recovered.
Syphilis causes termination of life
Published in Dinesh Kumar Jain, Homeopathy, 2022
Hahnemann mentioned the three causes of diseases: psora, syphilis, and sycosis. Causes of acute diseases are “Transient explosion of latent psora which spontaneously returns to its dormant state, if the acute disease were not of too violent a character and were soon quelled” (Hahnemann, 1921/1993, p. 160).
Right atrial reservoir strain and right ventricular strain improves in patients recovered from hospitalisation for non-severe COVID-19
Published in Acta Cardiologica, 2023
Mehmet Rasih Sonsoz, Gulden Guven, Ufuk Yildiz, Atilla Koyuncu, Ozlem Altuntas Aydin, Gokhan Kahveci
Retrospective analyses of the echocardiograms collected during hospitalisation and thus during acute disease (performed 24 h after admission) were compared with those collected at follow-up in our outpatient clinic (performed 6 [range, 5–7] months after discharge). Exams were performed during acute disease upon cardiology consultation for the differential diagnosis of acute heart failure, acute coronary syndrome and acute myocarditis. The patient’s cardiac symptoms were: shortness of breath in 58% (n = 25), chest pain in 40% (n = 17), and/or palpitations in 26% (n = 11). At follow-up, no patient showed signs and/or symptoms of acute infection, but 35% (n = 15) had shortness of breath, 21% (n = 9) had persistent chest pain, 42% (n = 18) had effort intolerance, and 23% (n = 10) had palpitations. Four of the patients (10%) were asymptomatic.
Inherited ADAMTS13 mutations associated with Thrombotic Thrombocytopenic Purpura: a short review and update
Published in Platelets, 2023
Zoe Markham-Lee, Neil V. Morgan, Jonas Emsley
Whilst in some cases immediate treatment for TTP may be required from birth, symptoms may first arise in adulthood, for example, after trauma such as infection or pregnancy. Stroke is an often-fatal manifestation of the disease in infants (frequency is 25–31% of patients with cTTP) [4]. Treatment of cTTP involves ADAMTS13 replacement either as fresh frozen plasma infusion or using an intermediate purity FVIII concentrate (8Y), pending licensing of recombinant ADAMTS13. Currently other, less invasive treatment options are still limited [5,6]. Immediate intervention dramatically decreases mortality rates and provides reduction of some symptoms such as headaches and abdominal pain in the majority of patients; as well as 80% reduction in TIA [7,8]. However, relapse is still common (40% in TTP and 69% in cTTP specifically) even when receiving treatment [8,9]. This suggests the treatment for acute disease is adequate but a search for long-term treatment would be beneficial.
Pathophysiology and mechanism of long COVID: a comprehensive review
Published in Annals of Medicine, 2022
D. Castanares-Zapatero, P. Chalon, L. Kohn, M. Dauvrin, J. Detollenaere, C. Maertens de Noordhout, C. Primus-de Jong, I. Cleemput, K. Van den Heede
We retrieved 54 articles that exclusively addressed hypotheses concerning potential underlying mechanisms of long COVID symptoms (see Table 1). Of these, 34 speculated on mechanisms that could specifically explain long COVID symptoms [10–43], whereas 18 focussed on organ injury developed during acute illness possibly resulting in persisting symptoms [44–61]. Concerning the latter, the suggested mechanism consisted of organ injury that complicated acute disease, possibly provoking persistent symptoms. The reported organ injuries were: stroke [46,52,55], myocardial infarction and fibrosis [41,43,49,51,55,57,59], acute encephalitis [43,50], neuromuscular disorders [40,41,43,46,48,56], renal failure [41,43,60,61], and hepatobiliary damages [43,44]. The other articles reported on endocrine disorders that were unrelated to organ damage, while including hypotheses on new-onset diabetes [62] and thyroid disorders [63].