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Cortical Blindness (Plus Anton-Babinski Syndrome, Blindsight, & Riddoch Syndrome)
Published in Alexander R. Toftness, Incredible Consequences of Brain Injury, 2023
Anton-Babinski syndrome, sometimes simply called Anton syndrome, is one of the most unusual disorders, full stop. This disorder occurs when blindness or partial blindness is combined with anosognosia, or the absence of knowledge about one's own disorder (see Anosognosia). Or, as Anton himself wrote it: “the symptom that we must emphasize in consideration of the following case was the fact that the patient was not conscious of her blindness” (Anton, 1899, translated in Forde & Wallesch, 2003, p. 203). In these cases, the person insists that they can see despite clear evidence that they cannot (Chen et al., 2015). For example, they may walk into walls, trip over furniture, and describe objects that they claim to see but in reality are not there (Maddula et al., 2009). These people may reportedly offer excuses for why they cannot seem to see, such as the room being too dark, or they may even try—and fail—to prove that they can see (Galetović et al., 2005).
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Published in Anton Sebastian, A Dictionary of the History of Medicine, 2018
Anton Syndrome Denial of visual disturbance or blindness. Described by an Austrian neurologist, G. Anton (1858–1933) in 1899. Fie graduated from the University of Prague in 1882 and succeeded Wernicke at Halle in 1905.
Perioperative Vision Loss after Non-Ocular Surgery
Published in Seminars in Ophthalmology, 2018
Bart Chwalisz, Aubrey L. Gilbert, John W. Gittinger
CVL is characterized by loss of vision with normal pupillary function and a normal funduscopic appearance. Central acuity is generally preserved unless bilateral damage has occurred. Occasionally, patients may be unaware of and deny cortical blindness (Anton syndrome). Complex and unusual higher-order disturbances of visual function may occur if the involved area is outside of the primary visual cortex, such as problems with recognition of color (central achromatopsia) and movement (akinetopsia), visual agnosia, and contralateral neglect. Additional neurological findings and decreased level of arousal may be present. CVL may be caused by embolic infarction of the posterior visual pathways and/or visual areas of the occipito-parietal cortex. Additional mechanisms of injury include watershed infarction, posterior reversible encephalopathy syndrome (PRES), and potentially various other uncommon pathologies such as cerebral hemorrhage and cortical venous sinus thrombosis.
Bilateral occipital lobe infarct neglect deficit (BLIND) syndrome
Published in Journal of Community Hospital Internal Medicine Perspectives, 2021
S Shanmugam, HL Haver, SM Knecht, R Rajjoub, O Ali, R Chow
Anton’s Syndrome has a distinct presentation, in which a patient is in denial of loss of vision, often confabulating visual perceptions in the setting of cortical blindness [1]. This phenomenon is referred to as visual anosognosia. Our patient presented with the classic description of Anton Syndrome. His imaging results are consistent with cortical blindness, likely cardioembolic in the setting of new onset atrial tachyarrhythmia. Paradoxically, the patient still perceived that his vision was preserved throughout his hospital course. Additionally, the initial presentation in which the patient described meeting a priest who picked him up and called the ambulance leads us to believe he may have been confabulating in the setting of his acute bilateral occipital infarcts.