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Rheumatology
Published in Paul Bentley, Ben Lovell, Memorizing Medicine, 2019
Buerger diseaseEpi: Middle-aged, Mediterranean men; smoking, HLA-B5 (B51)PC: CVA, PVD, Raynaud
B
Published in Anton Sebastian, A Dictionary of the History of Medicine, 2018
Buerger, Leo (1879–1943) American urologist of Viennese origin who graduated from the College of Physicians and Surgeons in 1901. He designed the Brown–Buerger cystoscope in 1909 which helped to irrigate the bladder during cystoscopy. See Buerger disease.
Diseases of Blood vessels
Published in P. Chopra, R. Ray, A. Saxena, Illustrated Textbook of Cardiovascular Pathology, 2013
Fig. 13.36b: Thromboangiitis obliterans (Buerger disease). Accompanying vein shows fibrin deposition and inflammation. Compare the involved segment with the uninvolved part of the vein which is seen in the upper part of the picture
Bullous pemphigoid in patients receiving peritoneal dialysis: a case series and a literature survey
Published in Renal Failure, 2021
Kohkichi Morimoto, Tadashi Yoshida, Naoki Washida, Kiyotaka Uchiyama, Takashin Nakayama, Hiroshi Itoh, Mototsugu Oya
A 72-year-old man with ESRD of unknown cause initiated PD in 2013. He could not choose HD, because he had Buerger disease and preparation of the vascular access on his upper limb was quite difficult. PD catheter was placed using a conventional method in February, and PD was initiated in April using 1.36% glucose solution for a daytime exchange and icodextrin solution for a night exchange. Four weeks after the induction of PD, he presented with pruritic eruptions on his hands. An icodextrin-induced allergic reaction was suspected. He stopped using it, but the pruritic skin lesion deteriorated to diffuse systemic blisters and erosions on his trunk and extremities in a week (Figure 1(A)). The laboratory data, PD condition, and PD prescriptions at the onset are shown in Table 1. The autoantibody for BP was positive (anti-BP180-NC16a antibody 143.9 U/mL [normal range <8.9 U/mL]), whereas the autoantibodies for pemphigus were negative (anti-Dsg1 antibody <3.0 U/mL and anti-Dsg3 antibody <3.0 U/mL). The results of skin biopsy showed subepidermal blister formation and eosinophil infiltration accompanied by the deposition of BP-specific autoantibody; the findings were compatible with BP. He was admitted to our hospital and treated with oral prednisolone (PSL) 1 mg/kg (60 mg) daily for 2 weeks to improve his skin lesions. Then, PSL was tapered (−10 mg/2 weeks) without recurrence. Three weeks after the initiation of PSL administration, an elevation in liver enzymes (aspartate transaminase 149 IU/L and alanine transaminase 145 IU/L), a low platelet count (5.0*104/µL), and atypical lymphocytes in drained peritoneal dialysate were observed. PCR analysis detected high levels of viral DNA (herpes simplex virus [HSV] 6 × 107 copies/mL and varicella-zoster virus [VZV] 2 × 102 copies/mL) in his blood. He was diagnosed with disseminated HSV and VZV infections. Acyclovir at a dosage of 3.5 mg/kg daily was administered, but his general condition deteriorated. Unfortunately, he also developed invasive pulmonary aspergillosis and septic disseminated intravascular coagulation caused by Enterococcus faecium. He died in July 2013.