Pancreatic disease

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Cellular Biology in Tissue Engineering

Published in Joseph W. Freeman, Debabrata Banerjee, Building Tissues, 2018

Extensive investigations with P-MSCs in pancreatic disease has led researchers to find that they possess the capacity to develop into insulin-producing cells.27 They also have the ability to produce islet-like cell clusters, which led to the restoration of normoglycemia cycles when transplanted into streptozotocin-induced diabetic Balb/C mice.28

Ensemble based biomarker identification on pancreatic ductal adenocarcinoma gene expressions

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Journal Information

Published in International Journal of Computers and Applications, 2021

Purbanka Pahari, Piyali Basak, Anasua Sarkar

We input all the quality IDs in DAVID device to think about practical explanations and KEGG (Kyoto Encyclopedia of Qualities and Genomes) pathway analysis. Some pathways related to cancer are found in the observation in Table 5. Further, this pathway examination additionally uncovers some more targets which oversee pancreatic disease such as focal adhesion, GnRH signaling pathway, pancreatic cancer pathway, renal cell carcinoma, chemokine flagging pathway and so forth. We find a few qualities among KEGG pathways, for example, FGS7, STAT5A, SPI1, CUL2, FAS MAP2K3, KRAS, SOS2, ADCY4, ADCY7, GRB2, AGNT2, AKT3RALBP1, TP53, SMAD3, CCND1, which are likewise in Pancreatic and other growth cell lines and in Table 5. Among these genes a few genes like AKT3, CHUK, KRAS, TP53 are now known as biomarkers for pancreatic growth [27].