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Work stress induced chronic insomnia in construction
Published in Imriyas Kamardeen, Work Stress Induced Chronic Diseases in Construction, 2021
Chronic insomnia can also be caused by psychiatric conditions such as chronic stress, anxiety, depression, bipolar disorder, obsessive compulsive disorder, hypomania, anorexia nervosa, schizophrenia, alcoholism and drug addiction (Riba 1993; Santos-Longhurst 2018). Paunio, Tuisku and Korhonen (2015) claimed that psychiatric conditions and insomnia have a bi-directional relationship and can become a vicious cycle.
Polyphenol Nanoformulations for Cancer Therapy: Role of Milk Components
Published in Lohith Kumar Dasarahally-Huligowda, Megh R. Goyal, Hafiz Ansar Rasul Suleria, Nanotechnology Applications in Dairy Science, 2019
Cancer can induce vicissitudes in human food consumption and/or dietary preferences. Anorexia, an advanced symptom in cancer patients reduce appetite and lead to cancer cachexia, while certain cancers can conversely induce increased appetite.125 Dietary interventions are frequently correlated with the occurrence and progression of malignant tumors and cancers in vitro, in animals and humans; however, integrated conceptual frameworks to interpret these changes are still vague. Mayne et al.76 emphasized to understand role of diet in malignant tumor development and progression.
Radiation
Published in Ronald Scott, of Industrial Hygiene, 2018
Above 100 rad, the cells that produce the epithelial lining of the gastrointestinal tract begin to show damage. Within a few hours a loss of appetite (anorexia) is displayed, then nausea at higher doses. By 200 rad vomiting is likely and by 250 rad diarrhea is probable.
Epigenotoxicity: a danger to the future life
Published in Journal of Environmental Science and Health, Part A, 2023
Farzaneh Kefayati, Atoosa Karimi Babaahmadi, Taraneh Mousavi, Mahshid Hodjat, Mohammad Abdollahi
Recent information on the relationship between epigenetics and eating disorders is limited. Most of the studies reviewed are of poor quality, so that no logical conclusions can be drawn yet. Most studies on the epigenetic impact of eating disorders have focused exclusively on DNA methylation. It was cross-sectional and centered mainly on females. For example, in primary studies of eating disorders, the global level of DNA methylation was measured and compared with healthy individuals, or DNA methylation was evaluated in selected candidate genes. Anorexia nervosa is a type of eating disorder. An experiment on female blood samples with anorexia nervosa showed hypomethylation of the SLC6A4 gene.[193]Extensive genome methods were also used to study epigenetic changes in EWAS.[194] DNA methylation was examined at the genome level in four groups: 75 women with active anorexia nervosa, 31 women recovered from anorexia nervosa, 41 women without eating disorders (NED), and a control group of 52. Most methylations were found on 58 genes involved in psychiatric status (serotonin receptor activity), immune and metabolic function, and nutrition (fat and glucose metabolism). Methylation levels at 64 sites associated with insulin function, genes regulating glutamate and serotonin activity were discovered. As well, increased methylation at 73 different sites related to inflammatory processes, immunity, olfactory, lipid metabolism, and glucose is associated with increased index of body mass.[109]