China
Africa
Explore chapters and articles related to this topic
Nutritional Deficiencies
Published in Philip B. Gorelick, Fernando D. Testai, Graeme J. Hankey, Joanna M. Wardlaw, Hankey's Clinical Neurology, 2020
Deepa Bhupali, Fernando D. Testai
Neurologic symptoms: Paresthesia and numbness in the feet and later the fingers.Lhermitte's symptom occasionally.Impaired memory.Anosmia.Reduced visual acuity.Diminished taste.Impaired manual dexterity.Lower limb weakness (corticospinal tract lesions and peripheral neuropathy).Unsteady gait.Impotence.
Syringomyelia/Syringobulbia
Published in K. Gupta, P. Carmichael, A. Zumla, 100 Short Cases for the MRCP, 2020
K. Gupta, P. Carmichael, A. Zumla
Syringomyelia is a condition characterized by the presence of a cavity in the spinal cord (most commonly in the cervical region). If the cavity is present in the brainstem, the term syringobulbia is applied. In most cases the symptoms appear between the ages of 25 to 35 years and included sensory impairment and weakness and wasting of the hands. Patients may have an unsteady gait with progressive stiffness. Light touch sensation is preserved but pain and temperature sensation are lost.
Test Paper 1
Published in Teck Yew Chin, Susan Cheng Shelmerdine, Akash Ganguly, Chinedum Anosike, Get Through, 2017
Teck Yew Chin, Susan Cheng Shelmerdine, Akash Ganguly, Chinedum Anosike
A 76-year-old man with known cerebral atrophy and dementia has been unsteady on his feet for the last 2–3 weeks following a fall down a flight of three steps. There is a small external cut in the parietal region of the scalp, but he was never brought to the A&E department. A CT brain has been performed to investigate his unsteady gait. It shows a large isodense subdural haemorrhage. All of the following are expected findings on the CT, except Fourth ventricle enlargementLateral ventricle compressionEffacement of the cortical sulciMidline shiftWhite matter buckling
A Novel Variant in CWF19L1 Gene in a Family with Late-Onset Autosomal Recessive Cerebellar Ataxia 17
Published in Neurological Research, 2021
Hussein Algahtani, Bader Shirah, Samah Almatrafi, Mohammad H. Al-Qahtani, Angham Abdulrahman Abdulkareem, Muhammad Imran Naseer
While performing a literature review, three previous publications have reported the clinical features and genetic analysis of four patients with novel variants in CWF19L1 (Table 1). In 2014, Burns et al [5]. reported two siblings born of consanguineous Turkish parents with non-progressive congenital cerebellar ataxia. They presented with motor delay, unsteady gait, and frequent falls. In their adolescent age, their physical examination showed florid cerebellar signs with hyperreflexia in the lower extremities. The IQ of one of their patients was low indicating cognitive impairment. Brain MRI of both siblings showed cerebellar hypoplasia predominantly affecting the vermis. Another study by Nguyen et al [6]. described a 10-year-old girl from a Dutch non-consanguineous family, who had dystonic movements in upper extremities, hypotonia, and cerebellar atrophy on MRI. Also, this patient had oculomotor apraxia and intellectual disability as additional features. Evers et al [7]. reported a 9-year-old boy from consanguineous Turkish parents who presented with motor developmental delay and unsteady gait with subsequent development of florid cerebellar symptoms and signs. Other interesting clinical features in this report were microcephaly and mildly impaired intellectual development. Sequential neuroimaging showed progressive cerebellar atrophy.
Sport simulation as a form of implicit motor training in a geriatric athlete after stroke: a case report
Published in Physiotherapy Theory and Practice, 2020
Sonia N. Young, William R. VanWye, Harvey W. Wallmann
The patient was a 76-year-old Caucasian English speaking male who presented to an outpatient physical therapy clinic 2 weeks after sustaining a right cerebrovascular accident (CVA) with left hemiparesis. He was hospitalized after onset of CVA for 3 days in an acute care hospital. While hospitalized, he received acute care physical therapy daily consisting of bed mobility, transfers, and gait training. After being discharged home from the hospital, he returned home with his spouse without home health nursing or physical therapy services. He was referred to physical therapy to evaluate and treat unsteady gait secondary to CVA. Diagnostic test results were later provided by the primary care physician’s office and indicated the presence of an embolic stroke; however, the exact location of the infarct was not provided. A carotid ultrasound was performed indicating a 57% stenosis in the right carotid artery and the presence of emboli on the right side causing left-sided symptoms. He was unable to drive a car at the time of the initial physical therapy visit and was brought to the clinic by his son. He was able to ambulate into the clinic with use of a single-point cane and close supervision to contact guard assist with no required orthotic device.
Unusual gait disorders: a phenomenological approach and classification
Published in Expert Review of Neurotherapeutics, 2019
Vijayashankar Paramanandam, Karlo J. Lizarraga, Derrick Soh, Musleh Algarni, Mohammad Rohani, Alfonso Fasano
The ‘cock gait’ is characterized by an extended trunk with flexed arms and strutting of the toes with heels not touching the floor as a result of a generalised dystonic posturing. This gait pattern has been reported in patients with manganese intoxication resulting in accumulation in the basal ganglia. Manganese accumulation could also result from recessive mutations of the SLC30A10 gene, chronic liver disease, prolonged parental nutrition and environmental exposures including drugs contaminated with manganese (e.g. ephedrone) [79]. The ‘peacock gait’ describes an excessive flexion of the hips and knees with plantar flexion of the foot during the swing phase of each step, which normalizes when supine and is well-described in primary and secondary dystonia [7]. As previously mentioned, the ‘pigeon gait’ describes ‘in-toeing’ while walking [5]. The ‘duck-like gait’ can be seen in tardive syndromes. This wide-based and unsteady gait with short stride length and mild ‘steppage’ phenomena can result from chorea and dystonia while walking [69].