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Cutaneous Manifestations of Sexually Transmitted Disease in the HIV-Positive Patient
Published in Clay J. Cockerell, Antoanella Calame, Cutaneous Manifestations of HIV Disease, 2012
Bryan Gammon, Antoanella Calame, Clay J. Cockerell
C. trachomatis gains entry to the body via skin breaks or microabrasions, although it can pass through epithelial cells of mucous membranes. It travels through the lymphatics and multiplies in the mononuclear phagocytic cells of the regional lymph nodes where it causes a characteristic disease pattern. LGV has been described as having three distinct stages. After an uncertain incubation period thought to be 1–2 weeks, a small painless papule or pustule appears at the site of inoculation, usually the glans or prepuce in men and the vulva or vaginal wall in women. The lesion usually erodes forming a small, asymptomatic shallow ulcer. If it develops, the ulcer heals without scarring within a week. Largely due to the location of the lesions in the genital area and the absence of symptoms, the initial lesion is often not noticed. In one study of 27 patients with advanced disease, only five patients recalled the presence of a primary lesion in the weeks preceding onset of systemic disease.107 Mucopurulent discharge from the urethra in males or the cervix in females should cause the clinician to suspect the diagnosis.
The ear
Published in Rogan J Corbridge, Essential ENT, 2011
This is usually due to infection of the middle mucosa and results in a mucopurulent discharge. This may also fill the ear canal if there is a hole in the drum. Infection can spread to structures closely related to the middle ear, and it is very important that one asks about symptoms such as vertigo, facial nerve weakness and headache.
Campylobacter concisus as a pathogen of primary canaliculitis: a case report
Published in Orbit, 2022
Yasuhiro Takahashi, Aric Vaidya, Hirohiko Kakizaki
On the first time, slit-lamp examination revealed a swelling in the medial part of the lower eyelid and a cystic change along the lacrimal canaliculus on the left side (Figure 1A). The left-lower punctum pouted without granulation tissue formation. The yellow mucopurulent discharge was expressed from the left-lower punctum by digital compression and was sent for the culture test. The culture was performed using sheep blood agar, chocolate agar, and Brucella HK agar media under anaerobic condition, aerobic condition, micro-aerophilic condition including highly concentrated hydrogen, and carbon dioxide. A culture test of the discharge showed Campylobacter concisus (1+), Gemella morbillorum (1+), Fusobacterium nucleatum (1+), and Porphyromonas gingivalis (2+) (Figure 1B). C. concisus grew well on the sheep blood agar medium under the aerophilic condition including highly concentrated hydrogen. All the microorganisms were found to have high drug sensitivity for ampicillin, clindamycin, cefmetazole, amoxicillin/clavulanate, imipenem, meropenem, and tazobactam/piperacillin.
Matrix Metalloproteinase-9 Point-of-Care Immunoassay after Dacryocystorhinostomy in Patients with Nasolacrimal Duct Obstruction
Published in Seminars in Ophthalmology, 2021
Primary acquired nasolacrimal duct (NLD) obstruction (PANDO) is a common acquired lacrimal disorder, which causes various symptoms such as continuous tearing, ocular surface discomfort, and recurrent conjunctivitis.1 The tear drainage system is very important for the maintenance of ocular surface health. During the normal functioning of tear secretion, tear flow washes away harmful chemicals and pathogens and keeps the cornea from being infected.1 If occlusion of the lacrimal duct occurs, it could cause intermittent or persistent epiphora and mucopurulent discharge. As a result, NLD obstruction can cause inflammation, and then accumulation of harmful materials and pathogens such as inflammatory mediators and bacteria in the conjunctiva sac, raising the risk for ocular surface disease.2
Acute dacryocystitis in pediatric patients and frequency of nasolacrimal duct patency
Published in Orbit, 2021
Abdulmajeed Alaboudi, Osama Al-Shaikh, Dalal Fatani, Adel H. Alsuhaibani
Congenital nasolacrimal duct obstruction (CNLDO) is the most common aetiology for acute dacryocystitis in children.2 It is characterized by persistent tearing and intermittent mucopurulent discharge from one or both eye(s). CNLDO has been reported to occur in 5–20% of newborns.3,4 The obstruction is due to the failure of nasolacrimal duct canalization most commonly at the membrane of Hasner distally within the nasolacrimal duct.5 Other etiologies of acquired nasolacrimal duct obstruction including sinusitis, post-traumatic nasolacrimal duct obstruction, and foreign body impaction have been reported.6,7Acute dacryocystitis in pediatric was also noted as a presenting feature of other systemic illnesses such as leukemia.8