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Histopathologic Correlations of Dermoscopic Structures
Published in Ashfaq A Marghoob, Ralph Braun, Natalia Jaimes, Atlas of Dermoscopy, 2023
Daniel Morgado-Carrasco, Constanza Riquelme-Mc Loughlin, Ralph P. Braun, Oriol Yélamos
Milia-like cysts are shiny whitish to yellowish round structures. They are more conspicuous when viewed with nonpolarized dermoscopy [19]. Histopathologically they correspond with intraepidermal keratin pseudocysts [67] (Figure 3.48). While the presence of multiple milia-like cysts is highly suggestive of SK, a few isolated milia cysts can also be observed in BCC, congenital nevi, papillomatous melanocytic nevi, and even melanoma [22].
Tissue Grafting Techniques
Published in Vineet Relhan, Vijay Kumar Garg, Sneha Ghunawat, Khushbu Mahajan, Comprehensive Textbook on Vitiligo, 2020
In the first 6 months, milia-like cysts can occur at the recipient site, especially on the face and neck. Many authors believe that milia develop due to remnants of epithelial cells following the dermabrasion, but a more plausible explanation for their development is occlusion of the sweat ducts where the outflow is blocked, in collaboration with rapid proliferation of the epithelial cells at the ends of the ducts [13]. A uniform dermabrasion and thorough cleansing of the dermabraded site before applying the graft can reduce this complication. Once developed, these can be managed easily by puncturing them with a fine needle and expressing out the contents. However, the authors have rarely encountered milia in clinical practice.
Epidermolysis bullosa acquisita
Published in Lionel Fry, Atlas of Bullous Diseases, 2020
The commonest areas to be involved are at the sites of trauma, i.e. the dorsa of the hands and feet (Figures 9.1 and 9.2) and the elbows. Blisters develop, and the surrounding skin is red and scaly. Scarring will eventually occur. Milia are a common feature in the healed skin. Nails may become dystrophic if the nail matrix is involved.
Management of resistant halo nevi
Published in Journal of Cosmetic and Laser Therapy, 2019
Sherif S Awad, Rasha TA Abdel Aziz, Sahar S Mohammed
Pigmentation of the graft was observed 2 weeks after the procedure. Depression at the site of the removed nevus was observed underneath the graft at the first month but disappeared completely afterward. Five cases started losing the pigment after 3 weeks, but with the maintenance of the NB-UVB sessions, complete repigmentation was achieved after the 3-month period. Milia were seen in five patients and were treated by simple evacuation with very good cosmetic outcome. In only one case, peri-graft halo persisted. No scarring developed in all cases either in the recipient or in the donor area. Further follow-up did not show any re-depigmentation in all cases, and satisfactory results could be traced up to 2 years postoperatively in some of the cases (Figures 2 and 3).
Complications and posttreatment care following invasive laser skin resurfacing: A review
Published in Journal of Cosmetic and Laser Therapy, 2018
Dan Li, Shi-Bin Lin, Biao Cheng
Typically, an open dressing refers to the direct application of an ointment or cream alone to the wound. Although inexpensive, convenient and easy for home care, this method may increase the risk of acne, milia and infection(50). Aquaphor is the most common ointment used for open dressings after LSR. Sarnoff compared the effects of the application of Aquaphor Healing Ointment (AHO) and Biafine Topical Emulsion (BTE) after fractional LSR. Twenty subjects were included in his double-blind, split-face study, and erythema, edema, epithelial confluence, and crusting/scabbing were assessed on days 2, 4, 7, and 14. The results showed that AHO was more effective in promoting wound healing than BTE(51). Tanzi et al. compared the effectiveness of a mucopolysaccharide-cartilage complex (MCC) healing ointment with that of Aquaphor. Their study showed that the MCC ointment, as a posttreatment care medication, was more effective than Aquaphor in reducing erythema, edema and erosion(52).
Emerging drugs for the treatment of epidermolysis bullosa
Published in Expert Opinion on Emerging Drugs, 2020
Matthias Titeux, Mathilde Bonnet des Claustres, Araksya Izmiryan, Helene Ragot, Alain Hovnanian
Dystrophic EB (DEB) is the second most common form of EB. Data from US registries indicate a prevalence of 3.3/1,000,000 [4]. DEB can be inherited either in an autosomal dominant or autosomal recessive pattern with extensive variability in the clinical phenotype [1]. Skin detachments and blisters, either spontaneous or secondary to minimal trauma, lead to milia formation, develop into chronic and extensive wounds with systemic complications especially in the generalized recessive forms. All mucous membranes (oral, esophageal, ocular, genital, anal) can also be affected. DEB is due to mutations in the COL7A1 gene, encoding type VII collagen (C7) the constituent of anchoring fibrils, which form essential structures for dermal-epidermal adhesion.