China
Africa
Explore chapters and articles related to this topic
What not to eat – how not to treat
Published in Vivienne Lo, Michael Stanley-Baker, Dolly Yang, Routledge Handbook of Chinese Medicine, 2022
Vivienne Lo, Luis F-B Junqueira
Some of the prohibitions are gathered together into specialist sections of the medical literature. This is most easily traced through the literature on women’s health in pregnancy and childbirth: what a woman should eat at different stages of pregnancy, what she should allow herself to see or listen to and how she should deport herself. One of the earliest manuscript collections devoted to medicine and matters pertaining to the gestating body was buried in Mawangdui tomb 3 in 168 BCE and is full of prohibitions to protect the development of the foetus: In the first month it is called ‘flowing in the form’. Food and drink must be finest: the sour boiled dish must be thoroughly cooked. Do not eat acrid or rank foods. This is called ‘initial fixture’. In the second month it first becomes lard. Do not eat acrid or stinking foods. The dwelling place must be still. For a boy there must be no exertion, lest the hundred joints all ail. This is called ‘first deposition’. In the third month it first becomes suet and has the appearance of a gourd. During this time it does not have a fixed configuration, and if exposed to things it transforms. For this reason lords, sires and great men must not employ dwarves. Do not observe monkeys. Do not eat onion and ginger; and do not eat a dish of boiled rabbit.(Taichan shu; trans. Harper 1998: 378–9)
Osteoarthritis
Published in Nicole M. Farmer, Andres Victor Ardisson Korat, Cooking for Health and Disease Prevention, 2022
Dietary patterns to be aware of for OA symptom management may also relate to development, not just prevention, of OA. Based on the pathophysiology evidence presented earlier, adverse dietary patterns may include high glycemic diets and diets that promote production of uric acid. Dietary factors related to uric acid exposure actually overlap with dietary patterns related to hyperglycemia. Added sugars that provide fructose either from sugar sweetened or naturally occurring fructose from fruit juices are linked to higher risk of uric acid formation and gout. Additionally, consumption of purine-rich animal foods, but not purine-rich vegetables, is linked to increased risk for gout. This suggests an adverse role for foods that contain stearic acid such as lard and tallow when present in a diet or meal that may lead to uric acid synthesis.
Diet and health
Published in Sally Robinson, Priorities for Health Promotion and Public Health, 2021
Heating fats to high temperatures, and re-heating them, encourages the development of unhealthy trans fats and saturated fats, as well as volatile aldehydes which are toxic (Harinageswara et al., 2010; Bhardwaj et al., 2016). Saturated animal fats such as goose fat, lard and butter are more chemically stable, and recommended for deep fat, extensive frying and baking. People in the UK need to eat more oily fish.
Comparative study of dietary fat: lard and sugar as a better obesity and metabolic syndrome mice model
Published in Archives of Physiology and Biochemistry, 2023
Victor Hugo Dantas Guimarães, Deborah de Farias Lelis, Luis Paulo Oliveira, Luciana Mendes Araújo Borém, Felipe Alberto Dantas Guimarães, Lucyana Conceição Farias, Alfredo Mauricio Batista de Paula, André Luiz Sena Guimarães, Sérgio Henrique Sousa Santos
In our study, we also observed an altered lipid profile in mice treated with high-fat/high-sugar diets, especially those fed with HBLS and HLHS. Increased total cholesterol, triglycerides, and VLDL-cholesterol levels were found mainly in mice treated with the aforementioned diets. Lard and butter usually have higher contents of saturated fatty acids (Almeida et al.2009), and although recent evidence has disregarded the harmful effects of these molecules on increasing cardiometabolic risk, the combination of fat and sugar augment the risk for metabolic disorders and is closely associated with adverse health outcomes (DiNicolantonio et al.2016). The lipid profile is connected to increased risk of cardiometabolic complications (Sanchez-Bayle et al.2008). Interestingly, studies report that a high-sugar diet may not only increase triglyceride levels but shift the molecules towards low-density particles (Krauss 2001). However, the literature is still controversial regarding which type of fat is “healthier”, as both (saturated and unsaturated) display benefits and harmful effects (DiNicolantonio and O'Keefe 2018).
The impact of obesity on brain iron levels and α-synuclein expression is regionally dependent
Published in Nutritional Neuroscience, 2019
Jian Han, Justin Plummer, Lumei Liu, Aria Byrd, Michael Aschner, Keith M. Erikson
Twenty male weanling (post-natal day 21) C57BL/6J mice (n = 20) were purchased from Jackson Laboratory (Bar Harbor, ME, USA). These mice were randomly divided into two dietary groups with 10 mice per group for 20 weeks. The control group was fed with a control-fat diet (CFD) (10% kcal from fat, D12450B; Research Diets) and the treatment group received a high-fat diet (HFD) (60% kcal from fat, D12492; Research Diets) according to an established obesity model diet.38,39 The source of the fat in the diets was lard. The diets included the same mineral mix (S10026; Research Diets), which provided 37 mg of ferric citrate per 4057 kcal. The similarity in iron content of the diets was confirmed by graphite furnace atomic absorption spectrometry (GFAAS) (Varian AA240, Varian, Inc., USA). Other major ingredients in the diets, expressed as g/kg diet, include casein (200 g/kg), l-cystine (3 g/kg), cornstarch (high fat: 452.2 g/kg, low fat: 72.8 g/kg), sucrose (172.8 g/kg), cellulose (50 g/kg), soybean Oil (25 g/kg), mineral mix S18708 (10 g/kg), and vitamin mix V10001 (10 g/kg). Food intake of the mice was recorded daily. Mice, housed individually in each cage, were provided with free access to diet and water 24 hours/day for 20 weeks. The housing environment was temperature controlled (25 ± 1°C) with automatic lights, which cycle off between 1800 and 600 hours. Before the initiation of the study, approval for all animal care and procedures was obtained from the University of North Carolina at Greensboro’s Animal Care and Use Committee.
Maternal obesity, diabetes during pregnancy and epigenetic mechanisms that influence the developmental origins of cardiometabolic disease in the offspring
Published in Critical Reviews in Clinical Laboratory Sciences, 2018
Prasoon Agarwal, Taylor S. Morriseau, Stephanie M. Kereliuk, Christine A. Doucette, Brandy A. Wicklow, Vernon W. Dolinsky
In contrast to the findings described above, Cannon et al. [163] did not observe differences in maternal high-fat diet-induced DNA methylation in the liver tissues of adult mouse (C57BL/6J) offspring. In these experiments, maternal obesity increased the body weight of female offspring fed a 58.0% kcal from fat diet post-weaning and also affected expression of genes involved in inflammation, cholesterol synthesis and RXR transcription factor activation in the liver tissues of the offspring, but had few effects on DNA methylation levels in liver [163]. Interestingly, these authors reported that the high-fat diet during pregnancy protected the male offspring from diet-induced obesity, in contrast with the findings of several other studies [102,124,164]. One possible reason for these conflicting results could be differences in the sources and composition of the fat in the diets (lard that is primarily comprised of long-chain saturated and monounsaturated fats vs. coconut oil primarily comprised of short-chain saturated fats).