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Anatomy for neurotrauma
Published in Hemanshu Prabhakar, Charu Mahajan, Indu Kapoor, Essentials of Anesthesia for Neurotrauma, 2018
Vasudha Singhal, Sarabpreet Singh
The internal cerebral vein is formed by the union of the thalamostriate veins (draining the caudate nucleus and the thalamus) and the choroidal veins (draining the choroid plexus of the third and fourth ventricles, along with the hippocampus, fornix, and corpus callosum).
Head
Published in Harold Ellis, Adrian Kendal Dixon, Bari M. Logan, David J. Bowden, Human Sectional Anatomy, 2017
Harold Ellis, Adrian Kendal Dixon, Bari M. Logan, David J. Bowden
This section passes through the posterior part of the opening in the tentorium cerebelli (27). Note the great cerebral vein (26), a short median vessel formed by the union of the two internal cerebral veins. It passes backwards to open into the anterior end of the straight sinus, which lies at the junction of the falx cerebri (9) with the tentorium cerebelli.
Cheryl
Published in Walter J. Hendelman, Peter Humphreys, Christopher R. Skinner, The Integrated Nervous System, 2017
Walter J. Hendelman, Peter Humphreys, Christopher R. Skinner
The system of veins that drain the deep structures of the brain emerges medially as the internal cerebral veins, one from each hemisphere (Figure 9.6b). These veins join in the midline in the region behind the diencephalon to form the great cerebral vein (of Galen). At this point, there is another exception to the rule of the formation of venous sinuses. A sinus is located in the midline, where the falx splays out laterally to form the tentorium. This is known as the straight sinus (Figure 9.6b and c). The great cerebral vein becomes continuous with the straight sinus, in the midline lying above the cerebellum. At this point, it is joined by the inferior sagittal sinus. At the back of skull, the straight sinus joins with the superior sagittal sinus. The venous sinuses now divide, and the blood flows into the transverse sinuses (Figure 9.6c and d). The venous blood exits the skull via the sigmoid sinus, becoming the internal jugular vein (Figure 9.6b and d), one on each side of the neck.
Review on the current treatment status of vein of Galen malformations and future directions in research and treatment
Published in Expert Review of Medical Devices, 2021
Panagiotis Primikiris, Georgios Hadjigeorgiou, Maria Tsamopoulou, Alessandra Biondi, Christina Iosif
In utero diagnosis and evaluation is very important and should nowadays include MRI imaging and to search for pseudo-feeders. The latter has been proven to be an important tool also after birth, not only in evaluating the brain parenchyma and ventricles and guiding therapeutics and timing of interventions but also in further elucidating angioarchitecture. Recent data have shown that the internal cerebral veins may communicate with the venous sac in some cases of VOGM [52]. In such cases occlusion of the VOGM venous sac can obstruct ICV drainage, unless there is a concurrent alternate drainage pathway. The detailed analysis of the angioarchitecture, using MRI protocols that include SWI sequences and 3DTOF, as well as CM or ASL MRA-MRV, may reduce procedure-related morbidity and mortality and enhance treatment planning, by providing further angioarchitectural data.
Importance of early recognition and management of delusional parasitosis
Published in Baylor University Medical Center Proceedings, 2022
Sabiha Armin, Genevieve LaPointe, Roy Jacob
She re-presented less than 2 weeks later after driving a nail into her head. CT of the head without contrast revealed a small intraaxial parenchymal hemorrhage along the right frontoparietal region, with the 9 cm metallic nail abutting the right internal cerebral vein (Figure 1a, 1b), with no evidence of vascular infarct or hemorrhage. She underwent a right temporal and bifrontoparietal craniectomy. After the procedure, the neurosurgeon showed the patient images of her brain to prove they were negative for parasites. Postoperatively, repeat imaging showed she had a new midline shift (Figure 1c). Psychiatry recommended indefinite treatment with aripiprazole 5 mg twice daily, sertraline 50 mg once daily, and regular visits with a psychiatrist. On several mental status exams, the patient was cooperative, with normal psychomotor activity. Her mood was euthymic and her speech normal in tone and volume, but mildly increased in rate. Her cognition and memory were intact; her thoughts were logical and goal-directed, with good insight and judgment. She denied suicidal/homicidal ideation and had improvement in delusions on aripiprazole, saying, “I’m not feeling any more movements in my head. [The surgeon] told me I didn’t have worms when they opened up my skull. I shouldn’t have done what I did.” Her husband stated he would help with her medication compliance and follow-up appointments. In the months leading to her cranioplasty, she experienced moderate headaches and continued with her levetiracetam for seizure prophylaxis. Through chart review, we found she stopped taking aripiprazole because of the side effect profile she read online regarding antipsychotics (although she denied experiencing metabolic or anticholinergic issues) and resumed having the original delusion.