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Paleolithic Diets
Published in Nathalie Bergeron, Patty W. Siri-Tarino, George A. Bray, Ronald M. Krauss, Nutrition and Cardiometabolic Health, 2017
Staffan Lindeberg, Maelán Fontes Villalba, Pedro Carrera-Bastos, Lynda Frassetto, Nathalie Bergeron, Patty W. Siri-Tarino, George A. Bray, Ronald M. Krauss
The extent to which alcohol could have been a regular part of human’s original environment is unknown (Kiple and Ornelas 2000). If storage vessels for alcohol were made of leather or plants in earlier prehistoric times, they have long since disappeared without a trace. Even without deliberate production of alcoholic beverages, a low-level dietary exposure to ethanol via ingestion of fermented fruit may have characterized our lineage of humans and human-like ancestors for about 40 million years (Smith 1999; Dudley 2002). For a comprehensive review of alcohol and cardiovascular disease, see Chapter 31.
Augmentation by resveratrol of the inhibitory effect of ethanol on platelet aggregation
Published in Platelets, 2023
Kazumi Ekawa, Mikio Marumo, Ichiro Wakabayashi
Light-to-moderate alcohol consumption has been shown to reduce the risk of ischemic heart disease in the general population [5,6]. The major reason for this preventive effect of alcohol on cardiovascular diseases is alcohol-induced elevation of blood HDL cholesterol level [7–9]. Blood coagulability and the risk of thrombotic diseases have been shown to be lower in drinkers than in nondrinkers [10]. In addition to lower levels of blood coagulation factors including fibrinogen, von Willebrand factor and coagulation factor VII in light-to-moderate drinkers than in nondrinkers [11], ethanol-induced inhibition of platelet aggregation has been suggested to be involved in the lower risk of cardiovascular disease in light-to-moderate drinkers than in nondrinkers [12]. Resveratrol has also been shown to inhibit platelet aggregation [13]. Therefore, both alcohol and resveratrol are thought to display antithrombotic effects in red wine drinkers. However, it remains to be clarified whether resveratrol has an additional effect on alcohol-induced inhibition of platelet aggregation.
Culturally Adapting Evidence on Dementia Prevention for Ethnically Diverse Communities: Lessons Learnt from co-design
Published in Clinical Gerontologist, 2023
Bianca Brijnath, Carolina Navarro Medel, Josefine Antoniades, Andrew S. Gilbert
Approximately 40% of dementia cases can be prevented or delayed by addressing 12 lifestyle factors: hearing loss, smoking, low levels of education, social isolation, inactivity, depression, traumatic brain injury, air pollution, alcohol, obesity, cardiovascular disease (CVD), and diabetes (Livingston et al., 2020). In the absence of a cure, the global consensus is that public health and health promotion efforts should focus on dementia prevention through addressing these modifiable factors (Livingston et al., 2020).
Supporting the role of penile trauma and micro-trauma in the etiology of Peyronie’s disease. Prospective observational study using the electronic microscope to examine two types of plaques
Published in The Aging Male, 2020
Aldo Franco De Rose, Guglielmo Mantica, Bruna Bocca, Alexander Szpytko, André Van der Merwe, Carlo Terrone
Despite numerous hypotheses, the etiopathogenesis of PD remains poorly understood [5–6]. The etiology seems to be multifactorial; specifically associated with risk factors such as smoking, alcohol use, cardiovascular disease, hypogonadism, diabetes mellitus and genitourinary inflammatory diseases [7–8].