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Sexual Dysfunction
Published in Peter Sagar, Andrew G. Hill, Charles H. Knowles, Stefan Post, Willem A. Bemelman, Patricia L. Roberts, Susan Galandiuk, John R.T. Monson, Michael R.B. Keighley, Norman S. Williams, Keighley & Williams’ Surgery of the Anus, Rectum and Colon, 2019
Somatic innervation is much less liable to injury, partly because the fibre lies beneath the pelvic parietal fascia and injury would involve dissection outside the normal planes of mesorectal excision rarely encountered other than in extended pelvic resection. They are, however, still liable to radiation injury (see below). The pudendal nerve may be injured during perineal surgery. The pudendal nerve, the pelvic autonomic nerves and sacral outflow may be damaged during traumatic vaginal delivery, particularly when excessive force is used or pressure exerted on the lower pelvic outlet.
Anatomy
Published in J. Richard Smith, Giuseppe Del Priore, Robert L. Coleman, John M. Monaghan, An Atlas of Gynecologic Oncology, 2018
Ernest F. Talarico, Jalid Sehouli, Giuseppe Del Priore, Werner Lichtenegger
The pudendal nerve is the main nerve of the perineum and the main sensory nerve of the external genitalia. Throughout its course it is accompanied by the pudendal artery. It exits the pelvis through the greater sciatic foramen between piriformis and coccygeus, then hooks around the ischial spine and the sacrospinous ligament, re-entering the perineum through the lesser sciatic foramen.
Perineal trauma
Published in David M. Luesley, Mark D. Kilby, Obstetrics & Gynaecology, 2016
Anal sphincter damage is mainly limited to first deliveries, whereas pudendal nerve damage can be cumulative. Pudendal nerve damage occurs during labour as the nerve becomes compressed and stretched. Delivery late in first stage or second stage by CS does not prevent this. It has also been shown that ultrasonographically visible anal sphincter defects can be demonstrated in women who were demonstrated to have an intact anal sphincter at the time of delivery. The mechanism for this late disruption is unclear. It may be related to infection or haematoma formation, or possibly to partial unrecognised sphincter ruptures.
Vulvodynia – an evolving disease
Published in Climacteric, 2022
Factors associated with vulvodynia onset include the following [16]:Chronic inflammatory stimulation of the vulva by recurrent candidiasis.Adverse response to hormones.Genetic predisposition to other painful disorders.Neurological disorders, especially multiple sclerosis.Significant emotional and psychological distress.Sleep disturbance or mood disorders.History of child abuse or molestation.Pelvic floor muscle overactivity and pudendal nerve entrapment.
Epidemiology of Peripheral Nerve Injuries in Sports, Exercise, and Recreation in the United States, 2009 – 2018
Published in The Physician and Sportsmedicine, 2021
Neill Y. Li, Gabriel I. Onor, Nicholas J. Lemme, Joseph A. Gil
This study also found a high incidence of football-related PNI in those 19 years and younger. The risk of PNI in football was also reported by Zuckerman et al. who used the High School Reporting Information Online (RIO) database and found the highest incidence of PNI occurred during football at 1.25/100,000 athlete-exposures followed by wrestling and baseball [35]. Cycling and swimming were also noted to have a relatively high incidence of PNI. In regards to cycling, pudendal nerve injury has been described given prolonged sitting on a bicycle seat [32,36]. Peripheral nerve-related injury was not noted though falls off the bike are likely high risk for such injury. In swimming, overuse shoulder injuries are reported to occur most frequently, as demonstrated in the collegiate population [37–39]. No specific nerve injuries were described, but a high incidence of overuse-related shoulder entrapment and impingement related pathologies may irritate the axillary, suprascapular, and subscapular nerves that innervate the deltoid and rotator cuff. In overuse or acute fashion, peripheral nerve injuries may occur in both individual and team sporting activities thus making it prudent to engage in proper technique, safety, and recovery practices to limit such occurrences.
Clinical effect of computed guided pudendal nerve block for patients with premature ejaculation: a pilot study
Published in Scandinavian Journal of Urology, 2020
Fouad Aoun, Georges Mjaess, Joseph Assaf, Anthony Kallas Chemaly, Tonine Younan, Simone Albisinni, Fabienne Absil, Thierry Roumeguère, Renaud Bollens
Anatomically, the pudendal nerve is a branch of the sacral plexus [19–21]. It emerges from the S2, S3 and S4 roots, then departs the pelvis from the greater sciatic foramen along with the sciatic roots, between the sacrospinal and the sacrotuberous ligament, and re-enters the pelvis via the lesser sciatic foramen [19–21]. At this level, it releases a superior hemorroidal branch and then cruises through a duplication of the obturator muscle’s fascia called ‘Alcock’s canal’. The pudendal nerve gives rise at the exit of Alcock’s canal to three main branches which are the inferior rectal branch, the perineal branch and the dorsal sensory nerve of the penis or clitoris [19–21]. The pudendal nerve entrapment consists of a compression of this nerve especially between sacrospinous and sacrotuberous ligaments (which constitutes the most common level of entrapment) or in Alcock’s canal [19–21].