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Nanotechnology and Delivery System for Bioactive Antibiofilm Dental Materials
Published in Mary Anne S. Melo, Designing Bioactive Polymeric Materials for Restorative Dentistry, 2020
Jin Xiao, Yuan Liu, Marlise I. Klein, Anna Nikikova, Yanfang Ren
Another aspect that has been associated with biofilm control and prevention of dental caries is the management of exposed dentin surface and dentin hypersensitivity. A recent work combined information from natural product research and nanotechnology to develop a versatile biomaterial, epigallocatechin-3-gallate-encapsulated nanohydroxyapatite/mesoporous silica nanoparticle (EGCG@nHAp@MSN), for therapeutic management of the dentin surface (Yu et al. 2017). This study demonstrated that the topical application of a slurry of the novel biomaterial was effective for dentinal tubule occlusion, hindering treated dentin more resistant against acid and abrasion. Moreover, EGCG@nHAp@MSN can continuously release EGCG, Ca, and P, significantly inhibiting S. mutans biofilm development on the dentin surface, is a promising material for clinical testing.
Facial pain
Published in Peter R Wilson, Paul J Watson, Jennifer A Haythornthwaite, Troels S Jensen, Clinical Pain Management, 2008
Peter Svensson, Lene Baad-Hansen
Dentine hypersensitivity is a common clinical problem where patients experience sharp or shooting pain in a tooth as a result of mechanical or thermal stimulation of the dentinal surface. Dentine hypersensitivity most often occurs in the cervical and root areas of the tooth.146 Cracked-tooth-syndrome refers to an incomplete fracture of a vital tooth that may extend into the pulp. The symptoms are a sharp pain evoked by chewing and is relieved by removing the pressure from the tooth.147 The pain is poorly localized and the radiographic examination does not reveal pathology.
Sealing effects of different Chinese herbal medicines on dentinal tubules: a scanning electron microscopic observation
Published in Ultrastructural Pathology, 2020
Yueheng Li, Jiao Chen, Yanxia Duan, Zhi Zhou
Dentin hypersensitivity is a common complaint in patients in the clinic. In 2008, through a comprehensive review of the literature, referring to the definition of dentin hypersensitivity proposed by the Canadian Expert Committee11 and Holland12, the Dentin Expert Group of Chinese Stomatological Association defined dentin hypersensitivity as follows: Exposed dentin produces short and sharp pain to external stimuli and cannot be attributed to dental defects or lesions caused by other specific causes. Typical stimuli include temperature stimuli, blowing stimuli, mechanical stimuli, or chemical stimuli.13 Dentin hypersensitivity is a special sensitive symptom of acid, softness, and pain when exposed to external stimuli, because wear, abrasion, and reparative dentin have not yet formed.
Prevalence of self-reported versus diagnosed dentinal hypersensitivity: a cross-sectional study and ROC curve analysis
Published in Acta Odontologica Scandinavica, 2019
Nayara Franciele Figueiredo Barroso, Polyana Matos Alcântara, Adriana Maria Botelho, Dhelfeson Willya Douglas-de-Oliveira, Patrícia Furtado Gonçalves, Olga Dumont Flecha
Several theories attempt to explain the mechanism of dentin hypersensitivity. The most accepted is the hydrodynamic theory, that postulates that the fluids of exposed tubules are disturbed by temperature, physical or osmotic changes and these changes and movements in dentin fluid flow stimulate baroreceptors present in the pulp and dentin, leading to neural discharge and resulting in pain [4].