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Bladder Tissue Engineering
Published in Gilson Khang, Handbook of Intelligent Scaffolds for Tissue Engineering and Regenerative Medicine, 2017
Bladder reconstruction, augmentation and complete replacement is indicated for a variety of clinical indications, ranging from bladder cancer, trauma, congenital defect (bladder exstrophy), neuropathic bladder, small-volume bladders, or inflammation.5 Current state-of-the-art methods rely on surgical techniques where autologous gastrointestinal tissues are harvested to augment bladders or create neobladders. The incompatibility of the gastrointestinal tissue (mostly ileum) with the bladder environment can lead to complications such as metabolic disorders, bladder perforation, stone formation, infections, hematuria-dysuria syndrome, and potential development of cancer due to metaplasia.6 The ideal outcome of bladder augmentation is a continent compliant bladder with self-control.7
List of Chemical Substances
Published in T.S.S. Dikshith, and Safety, 2016
Repeated exposure to high levels of p-chloronitrobenzene causes adverse health effects. The symptoms of toxicity include, but are not limited to, anoxia, unpleasant taste, anemia, methemoglobinemia, hematuria (blood in the urine), spleen, kidney, bone marrow changes, and reproductive effects. The target organs of p-chloronitrobenzene poisoning have been identified as the blood, liver, kidneys, cardiovascular system, spleen, bone marrow, and reproductive system.
Exertional rhabdomyolysis and acute kidney injury in endurance sports: A systematic review
Published in European Journal of Sport Science, 2021
Daniel Rojas-Valverde, Braulio Sánchez-Ureña, Jennifer Crowe, Rafael Timón, Guillermo J. Olcina
Since running is a moderate to intense prolonged exercise, there is evidence that both mechanical kidney trauma (Rojas-Valverde et al., 2019b) and muscle damage (Colombini et al., 2012; Rubio-Arias et al., 2019) could lead to conditions as proteinuria and hematuria. Likewise, evidence exists of cases of hemoglobinuria and myoglobinuria, which may be explained by the decreased eGFR, due to the temporal decrease of glomerular filtration rate.