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Pulmonary complications of bone-marrow and stem-cell transplantation
Published in Philippe Camus, Edward C Rosenow, Drug-induced and Iatrogenic Respiratory Disease, 2010
Bekele Afessa, Andrew D Badley, Steve G Peters
Presenting symptoms and signs of PVOD are non-specific.173 Radiographic findings include Kerley B lines, prominent central pulmonary arteries, and scattered patchy opacities.173,174 Unlike other forms of primary pulmonary hypertension, pleural effusions are common in PVOD.173 PFT demonstrates a decreased DLCO and restrictive ventilatory defect.4
Epidemiology, virology and clinical aspects of hantavirus infections: an overview
Published in International Journal of Environmental Health Research, 2022
Sima Singh, Arshid Numan, Dinesh Sharma, Rahul Shukla, Amit Alexander, Gaurav Kumar Jain, Farhan Jalees Ahmad, Prashant Kesharwani
Early symptoms of HCPS are similar to those of HFRS, including clinical and hematological signs identified during the febrile phase. HCPS is a severe acute condition often related to rapid on spring of respiratory collapse, leading to pulmonary, cardiogenic shock, and oedema. There are three steps of HCPS disorders: prodrome, cardiopulmonary, and recovery procedures. Nonspecific signs such as fever, headache, pain, and myalgia describe the prodrome for 1 to 5 days (Duchin et al. 1994). Thrombocytopenia could not have formed at an early stage of the prodrome, so in these conditions, a repeat platelet count may be warranted 8 to 24 hours later. Only thrombocytopenia is continuously present during the prodrome. This is an unspecific diagnostic test for HCPS. In the prodrome, platelet counts in HCPS patients have been reported to be fewer than 150.000 units in 80–95% of patients(Hallin et al. 1996). The onset of the cardiopulmonary stage follows increasing hematocrit and leukocyte count. History of residency or visiting a remote place, with or without established interaction with the rodent, and the existence of low platelets is an indication and warning of HCPS potential. Sadly, there are no clinical or experimental tests is available to determine the actual seriousness of the condition during the prodrome or early cardiopulmonary period. So, any patient suspected of HCPS should be taken to an intensive care unit. The beginning of the cardiorespiratory process requires 8–24 hours, with or without prior cardiac arrest (Mertz et al. 2006; Dvorscak and Czuchlewski 2014). Respiratory signs begin and respiratory findings of baseline crackles grow. Generally, radiography of the chest is increasingly evolving from ordinary to noncardiogenic pulmonary oedema- bilateral interstitial marks of Kerley B lines of typical size of the heart. It is with or without bilateral pleural effusions(Kitsutani et al. 1999).