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Food Fussiness in Early Childhood
Published in Mary Nolan, Shona Gore, Contemporary Issues in Perinatal Education, 2023
The neophobic stage starts at around 20 months and then gradually diminishes over the next three years (Nicklaus, 2009); eventually, most children will increase their range of accepted foods. Neophobia occurs in nearly all children, although some will be more neophobic than others. It is, to some extent, genetically determined – related both to parental neophobia and the child’s degree of sensory hypersensitivity (Cooke et al., 2007).
Nutrition and oral health
Published in Jackie Musgrave, Health and Wellbeing for Babies and Children, 2022
Children can be ‘picky’ eaters and show fear of trying new food, so called ‘neophobia’. This can cause anxiety and difficulties for adults, both parents and professionals who have responsibility for providing food to children. Tatlow-Golden (2019) points out that ‘educators can bear in mind that children are not to blame for fussiness and neophobia’ (p. 1). She goes on to explain that:fussiness and neophobia can be due to biological/developmental factors: (i) slower growth causing less eating; (ii) protective caution regarding new tastes at a time of first independent exploration; and (iii) increasing psychological autonomy. However, these behaviours can also be influenced by those around children, including their carers.
Preschool children: 1–4 years
Published in Judy More, Infant, Child and Adolescent Nutrition, 2021
Becoming wary of trying new foods is a normal stage in their development and is called food neophobia because ‘neophobia’ means fear of new. It is more evident in some toddlers while hardly noticeable in others. Parents begin to notice it around the time toddlers become independently mobile and are becoming more adept at investigating their environment. The fear of new foods is probably a survival mechanism to prevent mobile young toddlers from harming themselves through eating anything and everything: if they were to taste any interesting looking berry on a bush they could poison themselves.
Maternal overnutrition leads to cognitive and neurochemical abnormalities in C57BL/6 mice
Published in Nutritional Neuroscience, 2019
Christian Wolfrum, Daria Peleg-Raibstein
In the last decade, a link between maternal overnutrition/obesity and neurodevelopmental disorders in the progeny emerged.33 This association is not surprising, since the intrauterine environment has an important role in the normal development of the CNS of the unborn child.33 Animal studies have since shown that offspring born to overnourished or obese dams have decreased serotonergic30,34, dopaminergic29,35, and GABAergic signaling30 in brain regions, such as the mPFC, hippocampus, and striatum. These neuronal brain circuits have been associated with psychiatric disorders such as major depression, anxiety disorders, and cognitive impairments. We have previously observed that adult offspring born to HFD exposed mothers showed enhanced anxiety-like behaviors in the elevated plus maze and in the food neophobia tests while conditioned fear-response was intact. These behaviors were accompanied with increased levels of ventral hippocampal GABA(A) alpha2 receptor subunit, and 5-hydroxytryptamine 1A (5-HT1A) receptor.30
Centella asiatica prevents D-galactose-Induced cognitive deficits, oxidative stress and neurodegeneration in the adult rat brain
Published in Drug and Chemical Toxicology, 2022
Zeba Firdaus, Neha Singh, Santosh Kumar Prajapati, Sairam Krishnamurthy, Tryambak Deo Singh
The result of CAE treatment on SAS in the Y-maze test is shown in Figure 3(A). One-way ANOVA showed a significant difference in SAS between groups (F(2,12) = 10.13, p < 0.0026). Further, Newman-Keuls post-hoc analysis revealed that SAS of D-gal group (47.77 ± 10.22) was significantly lower than the Control (76.44 ± 16.68, p < 0.01) and D-gal + CAE group (82.06 ± 10.88, p < 0.01). Neophobia and anxiety in rats were assessed by observing their novel arm entries (Figure 3(B)) and coping behavior (Figure 3(C)) respectively. One-way ANOVA showed a significant difference in the novel arm entry between groups (F(2,12) = 13.38, p < 0.0009). Post-hoc analysis revealed that the percentage of novel arm entry in the D-gal group (26.43 ± 7.68) was significantly lower than Control (42.97 ± 5.22, p < 0.01) and D-gal + CAE group (40.93 ± 2.21, p < 0.01). Data analysis of coping behavior depicted a significant difference between groups (F(2,12) = 41.27, p < 0.0001). Further, post-hoc analysis revealed that the coping behavior of the D-gal group (18.03 ± 5.43) was significantly lower than Control (54.17 ± 7.06, p < 0.001) and D-gal + CAE group (45.59 ± 7.08, p < 0.001). The locomotor activity of animals was examined by monitoring the ambulation number in an open field test (Figure 3(D)). One-way ANOVA analysis revealed a significant difference in ambulation number between groups (F(2,12) = 17.21, p < 0.0003). The post-hoc analysis further showed that the ambulation number in the D-gal group (24.00 ± 6.89) was significantly lower than Control (44.20 ± 8.61, p < 0.001) and D-gal + CAE group (45.4 ± 2.07, p < 0.001). In all of these behavioral studies, differences between Control and D-gal + CAE group were insignificant (p > 0.05).
Feeding problems in children with autism spectrum disorders: a systematic review
Published in Speech, Language and Hearing, 2023
Rita Pinto-Silva, Ana Margarida Nunes Costa, Inês Tello-Rodrigues
In order to systematize all the relevant information, concepts were grouped into four main labels: food selectivity, mealtime behavioural problems, food neophobia and oral-motor impairment. Food neophobia is a term with significant divergence between authors. Several authors consider it to be a form of selectivity, while others look at it as a behavioural feature. This divergence is the reason why it has been analysed as a separate concept.