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Fatigue
Published in Carolyn Torkelson, Catherine Marienau, Beyond Menopause, 2023
Carolyn Torkelson, Catherine Marienau
You may find that taking one or two capsules of digestive enzymes 30 minutes before eating or with your meal helps with digestive issues. Also, there are natural digestive enzymes that aid in digestion such as pineapples, papayas, mangoes, and honey, as well as fermented foods like kefir, sauerkraut, and kimchi. In Ayurvedic medicine, it is not uncommon to begin the meal with a small glass of bitters to help your stomach release more gastric juices and prepare for digestion.
Cystic Fibrosis
Published in Charles Theisler, Adjuvant Medical Care, 2023
Fat-Soluble Vitamins: Cystic fibrosis frequently results in a deficiency of essential fatty acids and fat-soluble vitamins (A, D, E, and K) as well as fats, proteins, and calcium. The body cannot absorb them without digestive enzymes from the pancreas. For that reason, fat-soluble vitamins, calcium, and exocrine digestive enzymes are required as supplements.4
Towards the Importance of Fenugreek Proteins
Published in Dilip Ghosh, Prasad Thakurdesai, Fenugreek, 2022
Anti-nutritive compounds like hydrolase inhibitors affect digestive enzymes irreversibly. Trypsin and chymotrypsin inhibitors, as well as α-amylase inhibitors are known as the most important anti-nutritive compounds affecting protein digestibility (Duranti & Gius, 1997). Digestibility is a measure of protein hydrolysis and absorption of the liberated amino acids. Although an indication, digestibility could not thoroughly imply protein quality (Friedman, 1996). However, due to perquisite role of protein digestibility (i.e. susceptibility of peptide bonds to enzyme hydrolysis) on availability of amino acids and their integrity to oxidation, heat, etc., along with their absorption, it is worthy to measure protein digestibility under various conditions.
Nutrition Intake and Nutrition Status of Pancreatic Cancer Patients: Cross-Sectional and Longitudinal Analysis of a Randomized Controlled Exercise Intervention Study
Published in Nutrition and Cancer, 2022
Dorothea Clauss, Ingeborg Rötzer, Christine Tjaden, Thilo Hackert, Joachim Wiskemann, Karen Steindorf
The pancreas is closely involved in the metabolism of food and nutrients through the production of digestive enzymes and the secretion of hormones (insulin, glucagon) (1). Digestive enzymes are essential for the digestion and the utilization of nutrients. These metabolic processes can be affected by a disease of the pancreas (2). In patients with pancreatic cancer, the production of digestive enzymes is often reduced, disrupted, or no longer present, leading to maldigestion and malabsorption (3). The 52–88% of postoperative pancreatic cancer patients showed a medium–high risk of malnutrition (4). Malnutrition is associated with poorer prognosis (5). Many pancreatic cancer patients also suffer from symptoms including abdominal pain or nausea during eating, early satiety, vomiting and diarrhea, or constipation (6) that often result in an inadequate nutritional intake and absorption. In addition, many patients report a significant weight loss already at diagnosis (7, 8). Due to those physical and metabolic effects of the cancer and due to the effects of anticancer therapies, pancreatic cancer patients are at higher risk of malnutrition in the course of their disease history (9).
Identification of mite-specific eosinophils in the colon of patients with ulcerative colitis
Published in Autoimmunity, 2022
Shu-Wang Peng, Jiang-Ming Sheng, Bai-Sui Feng, Ke-Ping Peng, Gui-Xiang Tian, Cheng-Bai Liang, Ming-Hui Liu, Hai-Qing Xie, Qing Shu, Yan Li, Ping-Chang Yang
Previous reports indicate that HDM is detected in the intestinal samples [3]. Our recent studies also found that HDM-derived enolase protein induced neutrophilic inflammation in the mouse intestine [19]. The present data show that HDM also can be detected in the stool of both UC patients and NC subjects. The gastrointestinal tract is known to have various digestive enzymes. The event that HDM isolated from stool samples can be detected by Western blotting and ELISA demonstrate the bio activity of HDM. There are numerous immune cells in the colon tract. HDM can come into contact with immune cells to induce immune inflammation in the colon [3,19,20]. This reasoning is backed up by current data. By exposing to HDM in the culture or in the colon, Eos were activated by HDM, and released inflammatory mediators, MBP and ECP. As such, future studies may focus on the removal of HDM from the intestinal tract to reduce HDM-related inflammation in local tissues. This may be beneficial for patients sensitized to HDM, or/and prevent the development of HDM-specific immune inflammation [20].
Enhanced intestinal permeability and intestinal co-morbidities in heat strain: A review and case for autodigestion
Published in Temperature, 2021
Anthony A. Fung, Andy Zhou, Jennifer K. Vanos, Geert W. Schmid-Schönbein
A potential missing link between elevated intestinal permeability and fatal heat stroke is the leakage of digestive enzymes across the injured mucosal barrier, which has not yet been elucidated in the current literature on heat stress and strain in humans. Digestive enzymes are generated in the pancreas and released into the lumen of the upper (duodenal) segment of the small intestine. The enzymes are retained inside the lumen of the small intestine so that the mixture of food and digestive enzymes can continue to react while being carried via peristaltic motion along the intestine. The pancreatic proteases, amylases, lipases, and nucleases are powerful since they are concentrated, fully activated, and have relatively nonspecific activity. They can degrade most biomolecules, including one’s own intestine, and they generate inflammatory mediators during food degradation [88].