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Nutrition
Published in Jagdish M. Gupta, John Beveridge, MCQs in Paediatrics, 2020
Jagdish M. Gupta, John Beveridge
2.13. Which of the following statements is/are true about vitamin C?It is essential for the formation of collagen.It corrects transient tyrosinaemia in low-birth-weight infants.Formula-fed babies require vitamin C supplementation.Scorbutic rosary (due to vitamin C deficiency) is indistinguishable from rachitic rosary.Paucity of limb movements may be a presenting symptom of vitamin C deficiency.
Endocrinology
Published in Stephan Strobel, Lewis Spitz, Stephen D. Marks, Great Ormond Street Handbook of Paediatrics, 2019
Mehul Dattani, Catherine Peters
This condition usually presents with bone deformity, exhibiting different patterns depending on the child’s age at the onset of disease and the relative growth rate of different bones. In the first year of life, the most rapidly growing bones are the skull, the upper limbs and ribs. Rickets at this time presents with craniotabes, widening of the cranial sutures, frontal bossing, enlarged swollen epiphyses, particularly of the wrists, bulging of the costochondral joints (rachitic rosary) and a Harrison’s sulcus (Figs 13.60, 13.61).
Single best answer (SBA)
Published in Tristan Barrett, Nadeem Shaida, Ashley Shaw, Adrian K. Dixon, Radiology for Undergraduate Finals and Foundation Years, 2018
Tristan Barrett, Nadeem Shaida, Ashley Shaw, Adrian K. Dixon
Rickets is due to failure of bone mineralisation due to vitamin D deficiency; the equivalent condition in adults is termed osteomalacia. Other features include delayed closing of fontanelles, softening of cranial vault and the so-called rachitic rosary (enlargement of cartilage at the costochondral junction).
Rickets mimicker: a report of two cases of primary hyperparathyroidism in adolescence
Published in Journal of Endocrinology, Metabolism and Diabetes of South Africa, 2019
Imran M Paruk, Fraser J Pirie, Ayesha A Motala
Further inquiry revealed no family history of rickets or other medical conditions and he had a twin brother who was well. Clinical examination revealed proportionate short stature with a height of 131 cm and weight of 43 kg; Z-scores were −5.5 and −3.0 respectively. There were no dysmorphic features and he had delayed secondary sexual characteristics with a Tanner stage 2 appearance. Musculoskeletal examination revealed pectus carinatum, Harrison’s sulcus, kyphoscoliosis and rachitic rosary, suggestive of rickets. He had bilateral fixed flexion deformities of the knees with genu valgum on the right and he was unable to stand without assistance (Figure 1a–b). Systemic examination was normal. The working diagnosis was that of rickets, proportionate short stature and delayed puberty.
Rickets in a 6-year-old girl resulting in extreme deformities
Published in Paediatrics and International Child Health, 2018
On examination, she was pale and there was severe stunting and wasting. There were extension deformities of both wrists, and there was bowing of the long bones of the upper and lower limbs as well as a prominent rachitic rosary and Harrison’s sulcus in the chest wall (Figure 1). There was genu valgum of the legs and scoliosis of the thoracic spine.