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Tick Bites
Published in Charles Theisler, Adjuvant Medical Care, 2023
Removal: The tick can be grasped with clean, fine-tipped forceps or tweezers. The tick should be pulled out smoothly and gently without twisting. After the tick is removed, the bite area should be cleaned with rubbing alcohol or soap and water. For tick paralysis, tick removal is curative.2
Clinical Toxicology of Tick Bites
Published in Jürg Meier, Julian White, Handbook of: Clinical Toxicology of Animal Venoms and Poisons, 2017
The first rule of diagnosis of any envenoming is to actually think of envenoming as a cause of the patient’s symptoms and signs. This certainly applies to tick paralysis. Include tick paralysis in the differential diagnosis of anyone presenting with acute onset of ataxia, lower limb weakness, or more generalised paralysis, and in cases presenting in extremis following a convulsion, in whom a history of respiratory or speech difficulties or generalised weakness preceded the convulsion. This is particularly true for children. In seeking the diagnosis, do not lose sight of the immediate needs of the patient, and attend to these, particularly in regard to securing adequate respiratory function, if imperilled, and avoiding the possibility of aspiration of vomitus.
Ticks (order Ixodida)
Published in Eric S. Loker, Bruce V. Hofkin, Parasitology, 2015
Eric S. Loker, Bruce V. Hofkin
Pathology In addition to their role as vectors, ticks can cause anemia from heavy blood loss in severe infestations. Tick bites can additionally cause inflammation and ulceration, exacerbated by components of the tick’s saliva. Dermatitis in the ears (otoacariasis) is problematic for many animals. Toxic components in tick saliva can result in tick paralysis, when the bite is near the base of the skull. The paralysis is reversed when the tick is removed.
Multiple coinfections and Guillain Barré syndrome following outdoor travel to the American Northeast
Published in Baylor University Medical Center Proceedings, 2023
William Farrington, Farzam Farahani, Kevin Garrett Tayon, Jaclyn Rudzinski, Mark Feldman, Kartavya Sharma
A thick blood smear (Figure 1) demonstrated intraerythrocytic parasites with ringed, wisped, appliqué, and dual-organism morphology. Positive microbiology studies included Babesia microti polymerase chain reaction (PCR), IgM and IgG antibodies for Ehrlichia chaffeensis, IgM, IgG viral capsid antigen antibodies to Epstein Barr virus (EBV) and qualitative EBV PCR, and stool culture detecting Arcobacter butzleri. Stool culture was negative for Salmonella, Shigella, and Campylobacter species as well as Escherichia coli O157:H7. Cytomegalovirus serology was positive only for IgG antibodies. Testing for Borrelia burgdorferi, human immunodeficiency virus, and West Nile virus was negative. Given the lack of central nervous system involvement, meningismus, or cerebrospinal fluid pleocytosis, further testing for meningoencephalitis with an arbovirus panel or Ehrlichia chaffeensis PCR of the cerebrospinal fluid was not pursued. He did not have a past medical history of frequent infections or use of immunosuppressive medications. Tick paralysis was considered clinically but the patient’s timeline of symptomatic onset was too gradual, and sensory involvement was inconsistent with this diagnosis.