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Spinal Cord and Reflexes
Published in Nassir H. Sabah, Neuromuscular Fundamentals, 2020
Another ascending tract is the spinotectal tract, or the spinomesencephalic tract, that originates in the anterolateral parts of the spinal cord and terminates in the inferior and superior colliculi (Section 12.2.4.2). It is believed to be involved in inhibiting or controlling pain sensations. The spinothalamic tract, the spinoreticular tract, and the spinotectal tract constitute what is designated as the anterolateral system that carries information from the skin to the thalamus.
Specific Synonyms
Published in Terence R. Anthoney, Neuroanatomy and the Neurologic Exam, 2017
Antero2lateral fasciculus (A&V, p. 101–102 [including Fig. 7–2]) Antero-lateral spinothalamic tract2 (Bick, p. 158)Anterolateral system (K&S, p. 312)Spinal lemniscus (M&F, p. 30)Spinothalamic, spinotectal, and spinoreticular tracts (K&S, p. 312)Spinothalamic tract (B&K, p. 74)See, also, D: Spinal lemniscus and D: Spinothalamic tract.
Ernesto
Published in Walter J. Hendelman, Peter Humphreys, Christopher R. Skinner, The Integrated Nervous System, 2017
Walter J. Hendelman, Peter Humphreys, Christopher R. Skinner
Pain, temperature and crude touch pathways have already synapsed in the spinal cord and crossed near the point of entry and ascend as the lateral spinothalamic tract (Figure 2.6b; also called the anterolateral system). The tract ascends through the lateral medulla and continues through the lateral pons and midbrain to mingle with the ascending posterior column pathway (Figure 6.4).
Peripheral nerve stimulation: black, white and shades of grey
Published in British Journal of Neurosurgery, 2019
Viraat Harsh, Parijat Mishra, Preeti K Gond, Anil Kumar
The Gate Control hypothesis, which is currently the most widely accepted hypothesis, as proposed by Melzack and Wall1 published in Science 1965, explains the manner in which PNS works. The small diameter C fibres inhibit whereas the large diameter Aδ fibres excite the substantia gelatinosa, the overall inhibitory effect of which determines the nociceptive input and regulates its transmission to the cerebral cortex through the anterolateral system via the ventral posterior nuclei of the thalamus. The Aδ fibres inhibit the nociceptive input, thereby decreasing pain sensation. Aδ fibres having a lower threshold, get stimulated first, thus shutting the gate and inhibiting transmission of pain via the C fibres2. This implies that constant stimulation of Aδ fibres can diminish the transmission of pain at the level of spinal cord or medulla. This hypothesis was demonstrated by Ellrich and Lamp3 in 2005.
Dry needling as a novel intervention for cervicogenic somatosensory tinnitus: a case study
Published in Physiotherapy Theory and Practice, 2022
Aaron Womack, Raymond Butts, James Dunning
Additionally, Shore, Zhou, and Koehler (2007) reported that nociceptive projections to the cochlear nucleus (CN) appear to be lacking. The secondary neurons in the anterolateral system that mediate pain and temperature have not been reported to project to the auditory structures (Shore, Zhou, and Koehler, 2007). Furthermore, the CN contains very few nociceptive afferents from the spinal trigeminal nucleus (Shore, Zhou, and Koehler, 2007) Thus, alteration of somatosensory signaling, especially through the cervical muscles via DN, may be more significant than alteration of nociceptive signaling for treatment of CST. This may further help explain why the patient in the present case reported decreased tinnitus symptoms following manual but not electrical needle stimulation.
Pain management in multiple myeloma
Published in Expert Review of Quality of Life in Cancer Care, 2018
Pain is generally caused by stimulation of receptors specialized to detect noxious stimuli, called nociceptors. When they are activated, a signal is sent to the spinal cord, and then to the brain on pathways belonging to the anterolateral system, made up of three major tracts: spinothalamic, spinoreticular, and spinomesencephalic [7,8]. Fibers of the spinothalamic tract proceed to specific thalamic nucleus, then move to various cortical areas: the sensory cortex, responsible for accurate pain localization; the cingulate and insular cortex, involved in behavior, autonomic responses, emotions, and memories connected to pain [9–13].