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Hysteria
Published in Francis X. Dercum, Rest, Suggestion, 2019
Very frequently the patient associates in his mind loss of power with loss of feeling and in such cases, the part involved may present both palsy and anesthesia. Not infrequently, if there be rigidity, fixation or contracture, the patient reacts as though the part were painful. The sensitiveness, tenderness or pain—however it may be described—is typical in that it is superficial and presents the other characteristics of painful hyperesthesia which have already been described. It is frequently associated with the so-called "hysteric joints" (see p. 100).
Klippel–Trenaunay syndrome: Pain and psychosocial considerations
Published in Byung-Boong Lee, Peter Gloviczki, Francine Blei, Jovan N. Markovic, Vascular Malformations, 2019
Jamison Harvey, Megha M. Tollefson, Peter Gloviczki, David J. Driscoll
Neuropathic pain results from damage or dysfunction of neuronal pathways and is a shooting, burning, aching (or a combination) pain that is poorly responsive to conventional analgesics. The pain may be quite disabling and associated with hyperesthesia. Neuropathic pain can result from damage to nerves at the time of operation but can also result from effects of the venous abnormality on the nerve that shares the neurovascular bundle (e.g., pain associated with persistent sciatic vein). It may result from direct compression of the nerve and/or abnormal venous pressure of the nutrient vascular system of the nerve. The management of neuropathic pain is difficult, as such pain responds poorly to conventional analgesics and less well to opioids. Gabapentin or pregabalin are sometimes of benefit to neuropathic pain.
Physical symptoms
Published in Aurora Lassaletta, Ruth Clarke, The Invisible Brain Injury, 2019
Aurora Lassaletta, Susana Pajares
Alongside this loss of abdominal sensitivity, I have hypersensitivity in my back, hyperesthesia, which means I feel pain whenever I rest against a hard surface, like the floor, a firm mattress or a hard chair. It seems that the hyperesthesia relates to a particular sensitivity in fractured or seriously affected areas. Before I knew about this symptom, I had to attend several physiotherapy sessions and have another scan, since the doctors couldn’t explain the cause of this pain.
Surfer’s myelopathy: A review of etiology, pathogenesis, evaluation, and management
Published in The Journal of Spinal Cord Medicine, 2021
Jason Gandhi, Min Yea Lee, Gunjan Joshi, Sardar Ali Khan
Other signs include hypesthesia, hypoalgesia, and hyperesthesia. Between June 1998 and January 2003, nine cases of SM were reported, with an average patient age of 25, while exhibiting the aforementioned hallmark symptoms.1 In a reported case series of three patients in 2013 (24–31 years old; two male, one female),10 clinical manifestations also included bladder-bowel dysfunction with complete paraplegia (T9–12). Table 2 outlines the unique clinical presentation of gathered SM cases, a majority of which are due to surfing. Due to the likelihood of other conditions simulating SM, a proper differential diagnosis should be made while considering acute myelitis to broaden the spectrum (Table 3).6
Acute motor-sensory axonal neuropathy associated with systemic lupus erythematosus
Published in Baylor University Medical Center Proceedings, 2019
Smathorn Thakolwiboon, Amputch Karukote, Gyeongmo Sohn
A previously healthy 72-year-old white man presented with rapidly progressive bilateral ascending flaccid weakness and tingling sensation in all four extremities. He became quadriplegic within 3 days. Additionally, joint swelling, pain, tenderness, and morning stiffness were reported in both knees and finger joints. He denied dyspnea, voice change, and swallowing difficulty. He had no history of recent fever, diarrhea, or vaccination. At presentation, he was afebrile (98.8°F) and normotensive (126/65 mm Hg). He had no rash, hair loss, or oral ulcer. There was no superficial lymphadenopathy. Pain with passive movement and tenderness were noted in both knees and joints of the fingers. Neurological examination showed complete quadriplegia, hypotonia, and areflexia. Hyperesthesia was noted in all extremities. Cranial nerves were intact.
Early gain in pain reduction and hip function, but more complications following the direct anterior minimally invasive approach for total hip arthroplasty: a randomized trial of 100 patients with 5 years of follow up
Published in Acta Orthopaedica, 2018
B Harald Brismar, Ola Hallert, Anna Tedhamre, J Urban Lindgren
At 5-year follow up 7 surgical approach related complications had appeared in the DA group, but none in the DL group (p = 0.01). In the DA group 2 early dislocations were handled by closed reduction, neither of them recurred; 1 early deep infection was resolved following open irrigation, retention of the prosthesis, and antibiotics for 6 months; 1 patient dislocated 22 months postoperatively and was revised at 2 years and 9 months due to a pseudotumor; 1 patient developed hip pain 4 years postoperatively and was diagnosed with an iliopsoas cyst, revision was planned at 5 years; 1 patient had a late dislocation at 4 years and 7 months postoperatively and was handled by closed reduction, and, finally, 1 patient developed instability–subluxations at 1.5 years postoperatively, but did not find the disability severe enough to motivate revision surgery. Postoperative radiographs were analyzed in all patients with instability and all but one had cups positioned within Lewinnek’s safe zone (Lewinnek et al. 1978). That patient had a cup with 54 degrees of inclination and 16 degrees anteversion; he had 1 dislocation and no later recurrent instability problem. In the DL group, 1 patient developed hyperesthesia from the femoral cutaneous nerve of the opposite, unoperated leg, probably originating from pressure from the table support during surgery. 1 deep venous thrombosis occurred within 3 months in each group. 1 patient in each group had disturbed wound healing. Both healed uneventfully following treatment with oral antibiotics.