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Evaluating the Impact of Sleep Disruptions in Women through Automated Analysis
Published in Erick C. Jones, Supply Chain Engineering and Logistics Handbook, 2020
Shalini Gupta, Felicia Jefferson, Erick C. Jones
In the past decade, studies on OSA have identified associations between OSA and various cardiovascular complications, metabolic disturbances, and neuropsychologic deficits [39]. The effect of OSA on cardiovascular systems is a major topic in OSA studies where converging results have been found [46]. Atrial fibrillation is often observed after elective cardioversion in untreated OSA patients [52]. Although sleep apnea only happens during sleep, its effects extend into daytime body functioning [58]. OSA is recognized as a treatable cause of hypertension by the Seventh Joint National Committee of Hypertension guidelines [62]. Both enhanced sympathetic tone and genetic factors contribute to this causation between OSA and hypertension [64]. Conduction abnormalities such as sinoatrial block, sinus arrest, Atrioventricular (AV) conduction abnormalities, and asystole also occur during apnea in OSA patients [66]. The association between OSA and cerebrovascular events is also well studied [72]. OSA has been identified as a risk factor of cerebrovascular disease due to the high prevalence of pre-existing OSA among patients with stroke [77].
Tests for the identification of reflex syncope mechanism
Published in Expert Review of Medical Devices, 2023
Michele Brignole, Giulia Rivasi, Artur Fedorowski, Marcus Ståhlberg, Antonella Groppelli, Andrea Ungar
A reflex mechanism is likely when the following ECG findings are recorded during spontaneous syncope [18,54]: Progressive sinus bradycardia or initial sinus tachycardia followed by progressive sinus bradycardia until sinus arrest; orProgressive sinus bradycardia followed by AV block (and ventricular pause/s) with concomitant decrease in sinus rate; orRapid decrease in heart rate <40 bpm or >30% for >10 sSudden onset AV block (and ventricular pause/s) with constant P-P cycle, in absence of bundle branch block or structural heart disease (‘low adenosine’ idiopathic AV block [19])
Cryoballoon ablation beyond pulmonary vein isolation in the setting of persistent atrial fibrillation
Published in Expert Review of Medical Devices, 2022
Vincenzo Miraglia, Antonio Bisignani, Luigi Pannone, Saverio Iacopino, Gian-Battista Chierchia, Carlo de Asmundis
Another possible complication of the SVC isolation is the sinus node damage; in our experience [46] no permanent sinus node injury was observed; other authors have described few cases of transient SN injuries with sinus arrest and a lower atrial or junctional rhythm sometimes associated with extreme bradycardia. P-wave morphology and sinus node activity need to be monitored continuously and the application should be interrupted if sinus arrest or transient extreme bradycardia (<30 bpm) occurs. Usually, the sinus node recovers after cryoenergy interruption and a more distal SVC cryoapplication can successfully isolate SVC without further damage to sinus node [41].