China 
Africa 
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Plant Antioxidant Response During Abiotic Stress
Published in Hasanuzzaman Mirza, Nahar Kamrun, Fujita Masayuki, Oku Hirosuke, Tofazzal M. Islam, Approaches for Enhancing Abiotic Stress Tolerance in Plants, 2019
Deyvid Novaes Marques, Sávio Pinho dos Reis, Nicolle Louise Ferreira Barros, Liliane de Souza Conceição Tavares, Cláudia Regina Batista de Souza
Some NAC proteins are anchored in the plasma membrane or endoplasmic reticulum membrane (Liang et al., 2015). They are merged into transmembrane NAC protein subfamily named NTL (from NTM1-like). In response to abiotic stresses, their transport to the nucleus occurs after proteolytic cleavage (Seo et al., 2008). For example, MfNACsa (a lipid-anchored NACsa TF in Medicago falcata) is associated with membranes under unstressed conditions. Under drought stress, MfNACsa translocates to the nucleus through de-S-palmitoylation mediated by the thioesterase MtAPT1 (Wang et al., 2017a).
Transcriptome analysis of Takifugu obscurus liver in response to acute retene exposure
Published in Journal of Environmental Science and Health, Part A, 2020
Shulun Jiang, Di-an Fang, Dongpo Xu
Forkhead box class O (FoxO) is a nuclear protein subfamily that mediates the inhibitory actions of insulin or insulin-like growth factor on key genes. In turn, these inhibitory actions affect cell cycle regulation, energy metabolism, proteostasis, oxidative stress, apoptosis, and immunity.[9] Among the 19 FoxO signaling genes observed in this study, 18, including FOXO3, SMAD3, SMAD4, INSR, PI3K, and G1/S-specific cyclin D2 (CCND2), were significantly upregulated in response to RET exposure. The exception was tumor necrosis factor ligand superfamily member 6 (FASL) (Figure 8 and Table A3). FOXO3 is a transcriptional activator.[60]In vivo, FOXO3 reportedly promotes hepatic triglyceride accumulation, impairs glucose tolerance, and contributes to the upregulation of numerous lipogenic genes, such as GPAM.[48] A similar result has been reported in Caenorhabditis elegans[61] that FOXO3 stimulates lipid accumulation, so the abnormal expression of FOXO3 may impact lipid metabolism.