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Aquatic Plants Native to America
Published in Namrita Lall, Aquatic Plants, 2020
Bianca D. Fibrich, Jacqueline Maphutha, Carel B. Oosthuizen, Danielle Twilley, Khan-Van Ho, Chung-Ho Lin, Leszek P. Vincent, T. N. Shilpa, N. P. Deepika, B. Duraiswamy, S. P. Dhanabal, Suresh M. Kumar, Namrita Lall
Other genes that have been sequenced include the following: 1.5-bisphospate carboxylase small subunit, ATP synthase subunit I, ATPase subunit I, ATPase subunit III (atpH), fatty acid d6 desaturase, LHY homologue1, maturase K (matK), NPR1, NPR2, PbsI gene, phytochrome (PHYA), phytochrome C (PHYC), PsbK (psbK), psbK-psbI intergenic spacer, ribosomal protein S16, RNA polymerase beta subunit (rpoB), RNA polymerase C, and tRNA-Lys (trnK).
Individual conditions grouped according to the international nosology and classification of genetic skeletal disorders*
Published in Christine M Hall, Amaka C Offiah, Francesca Forzano, Mario Lituania, Michelle Fink, Deborah Krakow, Fetal and Perinatal Skeletal Dysplasias, 2012
Christine M Hall, Amaka C Offiah, Francesca Forzano, Mario Lituania, Michelle Fink, Deborah Krakow
Others: acromesomelic dysplasia Maroteaux type: severe acromesomelic shortening, joint laxity, autosomal recessive, vertebral anomalies (mainly kyphosis); caused by mutations of the gene NPR2; Limb/pelvis hypoplasia/aplasia syndrome (Al-Awadi/ Raas-Rothschild Syndrome) (p. 481).
Role of granulosa cell mitogen-activated protein kinase 3/1 in gonadotropin-mediated meiotic resumption from diplotene arrest of mammalian oocytes
Published in Growth Factors, 2018
Kankshi Sahu, Anumegha Gupta, Alka Sharma, Meenakshi Tiwari, Ashutosh N. Pandey, Shilpa Prasad, Pramod K. Yadav, Ajai K. Pandey, Tulsidas G. Shrivastav, Shail K. Chaube
LH dephosphorylates and inactivates guanylyl cyclase natriuretic peptide receptor 2 (NPR2) that results in the decrease of cGMP level on one hand and increase of cGMP hydrolysis through PDE5 in GCs on other hand (Egbert et al., 2018; Zhang et al., 2010). The epidermal growth factor receptor (EGFR) increases NPR2 activity and thereby cGMP level. However, mechanism by which EGFR reduces cGMP remains to be elucidated (Egbert et al., 2018). The decrease of cGMP level in GCs leads to reduced transfer of cGMP to the oocyte. Thus, a transient decrease of oocyte cGMP level triggers resumption of meiosis in follicular oocytes (Egbert et al., 2018; Robinson et al., 2012).
Investigating Mechanisms of Response or Resistance to Immune Checkpoint Inhibitors by Analyzing Cell-Cell Communications in Tumors Before and After Programmed Cell Death-1 (PD-1) Targeted Therapy: An Integrative Analysis Using Single-cell RNA and Bulk-RNA Sequencing Data
Published in OncoImmunology, 2021
Yi-Quan Jiang, Zi-Xian Wang, Ming Zhong, Lu-Jun Shen, Xue Han, Xuxiazi Zou, Xin-Yi Liu, Yi-Nan Deng, Yang Yang, Gui-Hua Chen, Wuguo Deng, Jin-Hua Huang
CCL19-CXCR3/CCRL2 interactions were relatively upregulated in responders versus nonresponders; the CCL19-CXCR3 interaction was mainly observed between DCs and all T cells/NK cells, and the CCL19-CCRL2 interaction was observed between DCs and macrophages. CCL4-SLC7A1/CCR8/CCR5 interactions were relatively stronger between activated CD8 T cells/macrophages and DCs/proliferative T cells/Tregs/tumor cells in responders than in nonresponders; among them, the CCL4-SLC7A1 interaction between activated CD8 T cells and DCs/proliferative T cells/tumor cells was drastically downregulated in nonresponders compared with responders, and the CCL4-CCR8 interaction between activated CD8 T cells to Tregs was drastically upregulated in responders compared with nonresponders. TNF-related ligand-receptor pairs, like TNF-TNFRSF1A and TNF-FAS, were largely upregulated between activated CD8 T cells and other cells in responders, while they were largely downregulated between activated CD8 T cells/macrophages and other cells in nonresponders. THBS1-a3b1 complex interactions were relatively downregulated between CAFs/endothelial cells/macrophages/myofibroblasts/tumor cells and tumor cells in nonresponders. NPR2-VEGFA/SEMA3C/PGF interactions between CAFs/DCs/endothelial cells/macrophages and other cells were upregulated in responders compared with nonresponders. LAIR1-LILRB4 interactions were downregulated between macrophages and DCs/macrophages in nonresponders. IL15-IL15RA interactions were relatively upregulated between DCs and CAFs/plasma cells in responders versus nonresponders. EREG-EGFR interactions between macrophages and CAFs/tumor cells and HBEGF-EGFR interactions between tumor cells and CAFs/tumor cells were upregulated in responders compared with nonresponders.
Neuroimaging and genetic characteristics of malformation of cortical development due to mTOR pathway dysregulation: clues for the epileptogenic lesions and indications for epilepsy surgery
Published in Expert Review of Neurotherapeutics, 2021
Nicola Specchio, Chiara Pepi, Luca De Palma, Marina Trivisano, Federico Vigevano, Paolo Curatolo
More recently, the application of next-generation sequencing (NGS) technologies allowed to study nuclear pedigrees not suitable for linkage analysis, or even sporadic cases, leading to the identification of four additional main genes: KCNT1 (Potassium Sodium-Activated Channel Subfamily T Member 1) [50], DEPDC5 (DEP Domain Containing 5) [51], NPRL2 (NPR2- like Protein) [52] and NPRL3 (Nitrogen Permease Regulator-like 3) [53].