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The Beneficial Effect of Omega-3 PUFA and L-Arginine on Endothelial Nitric Oxide (NO) Bioavailability
Published in Robert Fried, Richard M. Carlton, Flaxseed, 2023
Robert Fried, Richard M. Carlton
The vasa vasorum is a network of small blood vessels that supply the walls of large blood vessels, such as elastic arteries (e.g., the aorta) and large veins (e.g., the venae cavae). There are two sets of these vessels, one set outside, the other inside. Figure 3.3 depicts a segment of normal (pig) coronary artery showing the origin and spatial distribution of vasa vasorum.
Microsurgical Operations on Lymphatic Vessels in Man
Published in Waldemar L. Olszewski, CRC Handbook of Microsurgery, 2019
Technically, simpler is the procedure of invagination of the lymph vessel into the vein. Tension should be avoided. Sutures sealing the gap between the lymph vessel and vein should not go through the whole cross-section of the wall. In all types of LVS a special care should be taken not to deprive the lymph vessel of its blood supply (!). The lymphatic vasa vasorum can easily be seen under magnification.
Surgical Lympho-Venous Anastomoses for Treatment of Lymphedema
Published in Waldemar L. Olszewski, Lymph Stasis: Pathophysiology, Diagnosis and Treatment, 2019
According to our experience, this type of anastomosis should be performed in cases when there are dilated lymph vessels, easily dissected (chylous reflux, hyperplastic type of lymphedema), or when no suitable lymph nodes are available. Clinical and lymphangiography indications, as well as contraindications, are the same as for lymph node-vein shunts. Two types of operative techniques have been presented on Figures 3 and 4. One is an end-to-end anastomosis of dilated lymph vessel with a vein of the same caliber. To avoid a stricture at the site of anastomosis, a short tubing can be inserted into the lymph vessel and vein for the time of suturing and removed immediately before completion of the anastomosis through an incision in the proximal part of the vein. Technically more simple is the procedure of invagination of the lymph vessel into the vein. Tension should be avoided. Sutures sealing the gap between the lymph vessel and vein should not go through the whole cross section of the wall. In all types of LVS, a special care should be taken not to deprive the lymph vessel of its blood supply. The lymphatic vasa vasorum can easily be seen under magnification.
Interval growth in an ICA bifurcation aneurysm treated with balloon occlusion, and possible contribution of vasa vasorum hypertrophy
Published in British Journal of Neurosurgery, 2023
Muhammad Usman, Giles Critchley, Panayiotis Koumellis, Marius Poitelea
The serpiginous nature of the final vessel suggest hypertrophy of some of these interconnecting vessels (vasa vasorum) to reconstitute a vessel bypassing the blockage from the balloon within the vessel wall. An alternative explanation can be new vessels formed due to angiogenesis within the thrombus formed in the lumen of the occluded carotid, or even a combination of these new vessels and vasa vasorum in the vessel wall. Luminal recanalization without any vasa vasorum involvement would have to assume some balloon deflation which can happen but is unlikely to give a serpiginous appearance on angiography. Nonetheless, since there was no direct visualization of the pathological process in this case, our deductions are largely based on hypothesis drawn from previously existing bodies of literature. The end result of this was a relatively significant recanalization of the left internal carotid and this is likely to have contributed to the aneurysm enlargement.
Anti-neutrophil cytoplasmic antibody-positive vasculitis presenting with periaortitis and muscle vasculitis in a patient with chronic Chagas disease
Published in Scandinavian Journal of Rheumatology, 2020
V Garcia-Bustos, E Calabuig, J López-Aldeguer, P Moral Moral
Large-vessel involvement in ANCA-associated vasculitis is infrequent and fewer than 25 cases have been published to date. Periaortitis (1), stenosing aortitis (2), aneurysm, necrotizing arteritis, and even arterial rupture (3) have been described. The physiopathology is unknown, but histological analyses have demonstrated vasa vasorum vasculitis (2, 4), which may trigger the development of aortitis syndrome and eventually progress throughout the arterial wall, sometimes leading to death. Fibrinoid necrosis and granulomatous necrotizing vasculitis have also been found (5). Drug-induced cases after treatment with paclitaxel/carboplatin and bevacizumab combination chemotherapy (4) and etanercept (6) have been described. ANCA-positive vasculitis with large-vessel involvement can be associated with pulmonary capillaritis and glomerulonephritis (2, 7). As in our case, PET-CT has been previously used for diagnosing asymptomatic aortitis with inflammatory activity in ANCA-associated vasculitis. It has been suggested to be of prognostic value regarding late aortic complications (7). There is no standard treatment for this presentation. However, combined therapy with cyclophosphamide and methylprednisolone, tocilizumab or, as in our case, rituximab (8), has been successfully used.
Presence of inflammatory proteins S100A8 and S100A9 in a giant intracranial aneurysm after flow diverter treatment
Published in British Journal of Neurosurgery, 2019
Antonius M. de Korte, René Aquarius, Frederick J.A. Meijer, Peter van Lent, Hieronymus D. Boogaarts, Joost de Vries
Because the progressive vision loss did not respond to anti-inflammatory medication, we decided to decompress the optic chiasm by partial resection of the aneurysm. At surgery, compression of the optic chiasm and optic nerves was observed. The aneurysm was covered by vasa vasorum (Online Supplement 2). The superior part of the aneurysm was debulked and aneurysm tissue was collected from the wall and the intraluminal thrombus for histological examination. Vasa vasorum was coagulated to prevent bleeding. The patient recovered well and her visual fields and visual acuity was completely restored within 8 weeks after decompressive surgery (Online Supplement 1). Thrombus organization and aneurysm regression without diffusion restriction was seen on MRI 4 months after debulking. Patient consent regarding publication was obtained.