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Ventricular rhythms
Published in Andrew R Houghton, Making Sense of the ECG, 2019
Accelerated idioventricular rhythm is essentially a slow form of VT, with a heart rate of less than 120 beats/min (Figure 8.6). It occurs when an ectopic focus within the ventricles starts firing with a rate just higher than that of the sinoatrial node – this ventricular focus then takes over the cardiac rhythm.
Recognition of common arrhythmias
Published in John Edward Boland, David W. M. Muller, Interventional Cardiology and Cardiac Catheterisation, 2019
Nicholas P. Kerr, Rajesh N. Subbiah
Accelerated idioventricular rhythm may be observed following STEMI and typically occurs during the first 2 days. While it often occurs following reperfusion, it also occurs in patients without reperfusion, and is therefore not a reliable marker that reperfusion has occurred. Accelerated idioventricular rhythm does not affect prognosis and does not routinely require treatment.
ECG
Published in Fazal-I-Akbar Danish, Essential Lists of Differential Diagnoses for MRCP with diagnostic hints, 2017
Broad-complex (>3 small squares) tachycardia: often ventricular in origin:1 Narrow-complex tachycardia with aberrant conduction.2 VT.3 Accelerated idioventricular rhythm.4 Torsades de pointes.
A comprehensive review of cardiotoxic effects of selected plants
Published in Toxin Reviews, 2021
Akbar Anaeigoudari, Nahid Azdaki, Mohammad Reza Khazdair
In a report study, 4 males and 1 female, with ages from 49 to 78 years old, which one subject had ingested aconite leaves and 4 subjects had eaten aconite roots, showed ventricular tachycardia, premature ventricular contraction, accelerated idioventricular rhythm, and non-sustained ventricular tachycardia. Different alkaloids including; Aconitine, jesaconitine, mesaconitine and jesaconitine were detected in serum of patients (Fujita et al.2007).
Acute coronary syndrome from scombroid poisoning: a narrative review of case reports
Published in Clinical Toxicology, 2022
Cesare de Gregorio, Giuseppe Ferrazzo, Ioanna Koniari, Nicholas G. Kounis
Widened QRS occurred in a 44-year-old woman (case 6) who had accelerated idioventricular rhythm masquerading repolarization anomalies. In 2 patients (cases 8 and 9) there was transient ST elevation on the inferior leads, but case 8 was known to be an ischemic patient [34].
Successful Resuscitation from Refractory Ventricular Fibrillation by BLS Providers Employing Double Sequential External Defibrillation: A Case Report
Published in Prehospital Emergency Care, 2020
John Laird, Cesar Costa-Arbulu, Melissa Marighetto, Anna Grochal, Ian R. Drennan, Sheldon Cheskes
A 911 call was received by ambulance dispatch for an 83-year-old male in cardiac arrest in a private care facility. The cardiac arrest was witnessed by bystanders and the patient received continuous chest compressions until arrival of the fire department. The fire department continued chest compressions and analyzed the patient using an AED, resulting in a non-shockable initial rhythm. The first BLS ambulance arrived on the scene and attached a defibrillator monitor (Zoll E-SeriesR® defibrillator equipped with Real CPR HelpR® and CPR Stat-padz®). The crew immediately analyzed the cardiac rhythm resulting in VF and delivered an initial shock of 120 J. Two additional cycles of 2-minutes of CPR were performed. The patient remained in VF and was shocked at 150 J and 200 J as per local protocol. During the next cycle of CPR a second ambulance arrived on scene. It was recognized that the patient was in refractory VF and the defibrillation pads from the second defibrillator were placed in the anterior-posterior (AP) position without interrupting chest compressions to prepare for DSED (Figure 1). The initial set of pads were not removed or reapplied in preparation for DSED as they were properly spaced with good adherence to the patient. Both defibrillators were charged to 200 J and DSED was delivered by a single BLS provider pressing the shock buttons of both defibrillators in sequential fashion (Figure 2). A second DSED shock was delivered following an additional two minutes of CPR as the patient remained in VF (Figure 3). The second DSED resulted in an immediate return of spontaneous circulation (ROSC), confirmed by a palpable carotid pulse, a return of spontaneous respirations. Initial post-ROSC vital signs depicted a HR of 110 beats per minute in an accelerated idioventricular rhythm, a respiratory rate of 35 breaths per minute, blood pressure of 174/105 with an oxygen saturation of 100 percent, and an end tidal CO2 of 35 mmHg. The patient’s neurological status continued to improve during transport with an increase in GCS from 3 to 8 at the time of transfer of care at the receiving facility. A chronological timeline of events and interventions are reported in Table 1.