ExperimentaL Oral Medicine
Samuel Dreizen, Barnet M. Levy in Handbook of Experimental Stomatology, 2020
The experimental production of dental ankylosis in dogs by means of trauma was attempted by Parker et al.125 Conditions producing ankylosis in man often involve tissue injury, immobility, and lack of function. In the study, 16 dogs were anesthetized and the teeth examined radiographically. Splints were fabricated and cemented to immobilize selected teeth. Tissues investing the teeth were reflected and adjacent bone mechanically injured. The teeth were then ground out of occlusion. Although subsequent radiographic studies indicated that many of the treated teeth had become ankylosed, true ankylosis or continuity of bone with tooth substance was found in only one animal on histologic examination.
The History of Ankylosing Spondylitis
Barend J. van Royen, Ben A. C. Dijkmans in Ankylosing Spondylitis Diagnosis and Management, 2006
On the other hand, ankylosis of the spine can also be the result of noninflammatory chronic skeletal disorders as primary and secondary hyperostosis vertebralis. Primary hyperostosis is seen mainly in elderly cases and is considered to be a degenerative-metabolic disorder. In this group the sacroiliac joints are not fused. Secondary hyperostosis can be due to fluor- or vitamin A intoxication or to chronic use of retinoids. Ankylosis of a joint can be the result of direct local infection (e.g., toe), trauma, or immobilization. The characteristic of ankylosis due to infection, trauma, or immobilization is that in the majority of these cases only one joint will be involved.
The locomotor system
C. Simon Herrington in Muir's Textbook of Pathology, 2020
Ankylosing spondylitis is characterized by inflammation at the sites of insertion of ligaments into bone, the joint capsule and fibres of the annulus fibrosus of the intervertebral discs. The site of ligamentous insertion is the enthesis, and the resultant disease is known as enthesopathy. Inflammation is followed by fibrosis and ossification, particularly around intervertebral discs, with the formation of bridging spurs of bone (syndesmophytes). The synovitis histologically resembles that seen in rheumatoid arthritis. Bony ankylosis is more common than in rheumatoid arthritis, and may affect large joints, particularly the hips.
Affective Ankylosis and the Body in Fanon and Capécia
Published in Studies in Gender and Sexuality, 2018
Ankylosis, a rheumatological term referring to the inflammation that stiffens the joint and causes previously hinged bones to fuse, is just one in a series of conjoined metaphors that pepper Fanon’s works, all of which gesture to a figural hardening of motion wrought by the onset of colonization. For instance, Fanon explains that a culture, subject to racism, that was “once living and open to the future, becomes closed, fixed in the colonial status, caught in the yoke of oppression. Both present and mummified, it testifies against its members. It defines them in fact without appeal” (Fanon, 1967, p. 34). Other institutional apparatuses and relations of colonialism are at various points qualified as fixed, petrified, embalmed, inert, mummified, or tetanized, taking on the appearance and effect of the permanent, whereas racism itself assumes a fatal form of vitality. It “has not become encysted. Racism has not managed to harden. It has had to renew itself, adapt itself, to change its appearance. It has had to undergo the fate of the cultural whole that informed it” (1967, p. 32). The past is rendered contemporary in a deadening but not inanimate temporality. As we will show, affective ankylosis refers specifically to how this petrifying-effect takes place on the libidinal body to detemporalize or dehistoricize the subject.
Value of 3.0T MRI T2 mapping combined with SWI for the assessment of early lesions in hemophilic arthropathy
Published in Hematology, 2022
Lu Zhang, Shufang Wei, Jiajia Li, Pengming Wang, Ge Yinghui
Hemophilia is an X-sex chromosome recessive bleeding disorder. It is divided into two types, i.e. hemophilia A (factor VIII) and hemophilia B (factor IX), depending on the lack of clotting factors. The thickening of the synovial membrane stimulated by synovitis causes more joint bleeding, and the blood metabolite hemosiderin promotes the thickening of the synovial membrane, leading to the erosion of articular cartilage, and forming a vicious cycle of joint bleeding - synovitis - joint bleeding [7]. Furthermore, repeated synovitis with an accumulation of destructive enzymes and cytokines can destroy cartilage and subchondral bone [8]. In advanced stages, joint ankylosis and deformity may also occur, causing great pain and high disability rates. As the most common target joints are the knee [9], ankle [10], and elbow [11], in the present study, we focused on knee joints.
Tibial lengthening using a retrograde magnetically driven intramedullary lengthening device in 10 patients with preexisting ankle and hindfoot fusion
Published in Acta Orthopaedica, 2020
Bjoern Vogt, Robert Roedl, Georg Gosheger, Gregor Toporowski, Andrea Laufer, Christoph Theil, Jan Niklas Broeking, Adrien Frommer
We performed a retrospective analysis of patients treated with retrogradely implanted magnetically driven ILNs for tibial lengthening between 2015 and 2019. 10 patients (6 females) with preexisting ankle and hindfoot fusion (right = 8) were treated for correction of leg-length discrepancy (LLD). The underlying conditions were congenital (n = 9) and post tumor resection (n = 1) (Table 1). Ankle and hindfoot fusion was established by arthrodesing operations either precedingly in 8 patients or 1-stage with the ILN insertion in 1 individual. A congenital ankylosis was present in 1 patient (Figure 1). The mean preoperative LLD was 58 mm (36–80). 3 patients presented additional persistent angular deformities of the tibia (Nos 4, 6, and 9). The mean period between prior arthrodesis and intramedullary lengthening procedure was 23 months (5–62) (Table 1).
Related Knowledge Centers
- Ankylosing Spondylitis
- Inflammation
- Reactive Arthritis
- Synostosis
- Arthritis
- Osteoarthritis
- Vertebral Column
- Joint
- Injury
- Rheumatoid Arthritis